Stretching is a pleasant ritual for many people, myself included. It’s simple, it feels good, and we believe — or hope — that it prevents and treats injuries. For many others, athletes and couch potatoes alike, stretching is also a bit of a drag: a duty that weighs on the conscience, one more thing to make time for. And yet they still do it, counting on the benefits.
Can all these people be barking up the wrong tree? Yes. In fact, stretching is not an important part of fitness and wellness. Stretching’s primary goal — flexibility — does not have any clear value to anyone, not even most elite athletes, let alone the average fitness nut. It’s also easily achieved with other kinds of exercise that are more beneficial for fitness in other ways: strengthening can also improve flexibility.1 Most stretching is simply a waste of time, its reputation completely undeserved.
But stretching feels good!
You bet it does. Stretching is a waste of time for most people… unless you just enjoy it, of course.
I stretch almost every day — hamstrings, lumbar erector spinae, and especially the deep gluteals are my favourites. I do it because it feels nice, but I don’t believe the habit is doing much more for me than a daily back scratch. I am just as stiff and inflexible and full of “knots” as I have ever been. I play sports the same way with or without it. I still get just as sore, whether I stretch or not.
I am hardly the only person to notice that stretching may not be all it’s cracked up to be, and there’s plenty of science on this.2 This free online book — yes, it’s a short book, about 34,000 — is a readable review of the key arguments and concepts and some of the most interesting evidence. Warning: you may not enjoy stretching quite so much once you realize how many myths there are about its benefits.
Flexibility has been researched for over 100 years. Its track record is unimpressive, particularly when viewed in light of other components of physical fitness. Flexibility lacks predictive and concurrent validity value with meaningful health and performance outcomes. Consequently, it should be retired as a major component of fitness.
Nuzzo, 2020, Sports Medicine
This book is not just about the inadequacies of static stretching: just lengthening muscles and then holding still for a while. Many stretching fans are happy to join me in criticizing simple, old-school static stretching. They are happy to do that because most of them decided long ago that some other method of stretching actually does work, and that’s where their allegiance lies.
Unfortunately, there is no clear evidence that any method of stretching is a clear winner for any important therapeutic goal. All stretching is either a trivial variation on one of the basic themes, or too different to really be considered stretching in the first place (e.g. there are other “active ingredients,” like in yoga). Here’s an introduction to the members of the stretching family:
- Static Stretching — Just elongating muscles for a while, mainly varying in the intensity and duration.
- Neurodynamic Stretching — A noteworthy sub-type of static stretching devoted to stretching nerve tissue.
- Dynamic Stretching — Stretching with movement. Essentially all branded methods of stretching are some variation of dynamic stretching. Some major sub-types:
- Ballistic Stretching — Using momentum to repeatedly, briefly push past the end of your comfortable range of motion.
- Contract-relax Stretching — Alternating stretches with contractions. Also rather grandiosely known as “proprioceptive neuromuscular facilitation” (PNF).
- Dynamic Joint Mobility Drills — Rhythmic, repetitive movement through a full range of motion than usual (i.e. swinging your arms in a circle or walking lunges).
- Mixed — Complex exercise practices that include some stretching, but also much else. Yoga is the ultimate example, but virtually any activity that by nature requires range of motion — like martial arts — can be considered a complex dynamic stretching exercise.
- Pandiculation — Spontaneous brief stretches, often combined with yawning, that most vertebrate animals seem to do routinely and reflexively after a period of stillness. Human stretching seems to be an exaggeration of this basic feature of physiology.
So what is “advanced” stretching?
There are no special members of the stretching family. For instance, PNF is nothing fancy, despite the high-falutin’ name: the contract-relax approach just adds contraction.3 And it doesn’t increase flexibility any more than static stretching.456 At best, PNF might have subtle advantages for specific situations.
PNF is not so advanced after all. And neither is anything else that has ever been touted as an advanced stretching method.
There have been countless trademarked stretching methods, probably thousands of them over the decades, most of them distinguished more by their marketing than by the details, all claiming to be advanced. For all that variety, they are all tediously similar, mostly just variations on dynamic stretching, which all integrate so much movement that they are more about movement than “stretching.” To the extent that they work, they probably work because they are just exercise — and would still work if you stripped out the stretching.
This is a fine example of an advertisement for an advanced stretching method that not only improves flexibility quickly, but helps men “strength the entire core with strain” & “burn a ton of calories.” How plausible is it that there is a secret method of exercise that can improve cardio, strength & flexibility quickly & without strain? Even if it worked, how could it possibly be just “stretching”? Also note the absurd level of engagement…on an advertisement.
So “advanced” it’s not even stretching anymore. A few of the most distinctive have earned their own identity only because they are thoroughly mixed up with other concepts (again, yoga being the most obvious example). They have fallen so far from the stretching tree that they cannot really be considered stretching anymore. There are only so many things that you can change about stretching before it really becomes something else.
I don’t really think there is any such thing as “advanced” stretching — just basic stretching with delusions of grandeur. This book is about everything that can reasonably be considered stretching, which are all pretty similar — and it is not about anything else. And the operational definition of stretching for this book is:
Stretching is any exercise that involves elongating muscles to the point of feeling significant tension for at least several seconds.
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I will begin tipping over stretching’s sacred cows with this extremely controversial statement: anatomy has limits. Strict limits. An owl can rotate its head as much as 270° and you can’t! Owl spines and people spines are different.
Although anatomy is amazingly variable (for some interesting examples, see You Might Just Be Weird) it still works about the same for most people, and there are anatomical limits on all stretches. Some more than others.
Tensile force can not be applied equally to all muscles. Most of us will hit the end of the natural range of motion of the joint long before we’ve stretched anywhere near as hard as you can stretch other muscles. In other words, some muscles are just biomechanically awkward to stretch. I call them “the unstretchables” — a bit of hyperbole, but true in spirit. Although these muscles can be elongated, they can’t be elongated enough to create the satisfying sensation of good stretch.
There are several important muscles and muscle groups that are mechanically impossible to stretch much, including ones (like the quadriceps) that people think they are stretching.7 Even if stretching actually had the benefits that people believe in — which it clearly does not — those benefits would still not actually be available for large areas of our bodies.
This biomechanical reality is spread over the whole topic of stretching like a large, wet blanket. Whatever stretching can do for us, if anything, it has do it within the significant constraints of our anatomy, like a suntan lotion that we can’t apply to our own back. As you read, bear this broad limitation in mind.
For much more detail, see: The Unstretchables: Eleven muscles you can’t actually stretch hard (but wish you could)
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Athletes stretch mostly to warm-up, prevent and treat injuries, and to boost performance mainly through flexibility. Only one of those popular reasons for stretching has ever held up to scientific scrutiny, and even that one is only half true: stretching will indeed make most people more flexible, if you work hard enough at it, but flexibility is mostly worthless to the average athlete, and even most elite ones.
Stretching has a place of honour in athletics, sports medicine, and fitness — it is an activity everyone loves to love — and yet it is arguably the most over-rated thing you can do with a body.
But for the science of stretching in sports and fitness… just keep scrolling.
What a sensible article, and about time somebody exploded the stretching myth! I remember as a schoolboy in South Africa forty years ago always being told to run slowly to warm up for our various rugby, cricket, and soccer games — nobody ever told us to stretch, and over the past ten or so years I’ve been puzzled to see this come in as dogma. As a runner of marathons for years and a GP with injured patients, I’ve never been able to figure out how on earth stretching the heck out of muscles, ligaments, and nerves could (a) warm them up or (b) do the slightest bit of good, and have sometimes been given “the jaundiced eye” when I’ve suggested such to my patients.
Peter Houghton, MD, Vancouver (reader feedback)
An overview of the five reasons (plus one bonus reason)
When challenged, stretching enthusiasts — both casual and hardcore — have a surprisingly hard time explaining why they are stretching. Everyone just “knows” that it’s a good thing, and they haven’t really thought about why. It’s dogma, practically a religion.
When pressed for reasons, most people will cough up a few predictable stretching goals. Here are the four hopeful reasons for stretching that I hear every day:8
- flexibility, of course
- warm up and injury prevention
- prevention/treatment of exercise soreness
- treatment of sports injuries and chronic pain
And a fifth which I only hear occasionally, but it’s still out there:
- “performance enhancement” (e.g. faster sprinting)
All of these overlapping goals for stretching have serious problems. Either they have long ago been proven to be impossible,9 or they never made sense to begin with, or both, or worse. Certainly none of them is a slam dunk. They will all be critically analyzed below.
Bonus reason to stretch!
It feels good, obviously. This is the neglected bonus reason for stretching I can get behind.
You cannot “warm up” your muscles by stretching them: it’s like trying to cook a steak by pulling on it. Instead, the best way to warm up is probably to start by doing a kinder, gentler version of the activity you have in mind (i.e. walk before you run). The metabolic activity involved in muscle contraction does literally warm up your muscles, an inevitable side effect of all the activity that actually makes warm up effective for injury prevention.
Nothing about static stretching is more clear than this failure. Your basic quick (static) stretch warmup is one of the most studied topics in all of musculoskeletal health care and exercise science. For instance, a huge 2011 review of all the research found “overwhelming evidence that stretch durations of 30-45 seconds … imparted no significant effect” and even some evidence of harm.10
Um, harm? Slight harm, yes: stretching may actually reduce performance. Probably not much, but also probably not what anyone wants. More on this shortly.
Metaphorically, “warming up” also refers to readiness for activity or body awareness. You are “warm” in this sense when you are neurologically responsive and coordinated: when your reflexes are sensitive and some adrenalin is pumping. Warmup for its own sake (i.e., without following it up with more intense exercise) is fairly pointless — the goal is to prevent injury and enhance performance. And those goals may be realistic. For instance, research has shown that a warmup routine focused on these goals actually does provide decent insurance against the number and severity of both accidents and over-use injuries.1112
So, warmups in this second sense is probably helpful … but does stretching warm you up in this sense? No, probably not much — certainly no more than a bunch of other exercises you could do — and quite possibly not at all. One of the most-studied warmup regimens (including one of the studies just cited), FIFA’s “The 11+” programme, notably does not include stretching.13 The most compelling evidence that stretching doesn’t warm you up is the evidence that shows that it doesn’t prevent injury or enhance performance (discussed below)… but it can actually cause some injuries, and might even impair some kinds of performance! Much more on those backfire effects in the sections ahead.
Static stretch is somewhat stimulating to tissue, but in ways that are quite different from most actual activities.
A large study of girls’ soccer teams showed warming up can cut injury rates by about a third. Notably, the warmup that was studied, FIFA’s “11+” warmup, did not include stretching!
Because of all this, stretching to warm up does not even qualify as “official” exercise dogma anymore — most professionals actually gave up on it many years ago, and it is passé even in the opinion of a great many more informed joggers and weekend warriors. It simply doesn’t work, and it’s hard to imagine a common fitness practice more thoroughly contradicted by the evidence and by many professionals. And yet …
And yet I still see it all the time in the wild. I live and play on Vancouver’s famous “sea wall” — one of the best and most popular running routes in the world. And so I can constantly observe runners in their natural habitat, doing what runners do. A great many of them participating in structured training programs and running groups, instructed and led by experts and coaches… and they stretch to warm up. In droves. So despite the evolution of professional opinion, this practice clearly still needs to be discussed and debunked.
I am also an avid ultimate player,14 and so I also routinely see seriously athletic people stretching to warm up (and often hear their justifications for it, too, which are always bog standard stretching dogma).
Clearly there are still far too many people out there stretching before they run and play sports, trying to “warm up” almost exclusively by standing still and elongating muscles!
Once again, the best way to prepare for an activity is probably just to start it slowly.
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Another extremely popular idea about stretching is that it prevents a specific type of soreness: the deep soreness that follows a hard workout. That phenomenon is called “delayed-onset muscle soreness” (DOMS) or sometimes just “post-exercise soreness.” Many people believe that stretching can help DOMS. Some seem to believe it like it’s their religion, and it’s amazing how determined people can be to ignore evidence that contradicts it.15
Recently (2016), decades after this faith first got entrenched, science finally coughed up a specific reason why it might be based on something: evidence that stretching reduces inflammation in connective tissue.16 (Exercise in general is anti-inflammatory,17 and maybe this is partly why.) Surely this is relevant! It sure sounds like a great science factoid to explain why people think stretching helps soreness after exercise. Practically a slam dunk! It is interesting evidence for sure, and I’ll return to it later.
But there are many problems with leaping to that conclusion.18 It’s a classic mistake to assume that a scrap of biological relevance translates into a clear benefit in the real world. Unfortunately, the evidence strongly suggests that stretching does not prevent DOMS. Many studies have shown that nothing short of amputation can prevent DOMS192021 — and certainly not stretching.22 Whatever effect stretching has on inflammation in connective tissue, it does not add up to a DOMS cure.
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The commonly accepted idea that increased ROM and stretching prior to activity prevents injuries has been challenged and found to be on the shakiest of scientific foundations, or to come from such a paucity of data that no reasonable conclusions can be drawn.
Flexibility, by William Sands, p. 389
According to the evidence, stretching probably does not prevent injury. As I mentioned above, this has been suggested by a combination of recent literature reviews and large clinical studies, some of which I have already cited. Here’s some more.
In 2005, the Clinical Journal of Sports Medicine published a review of the scientific evidence to date, and found that the (admittedly limited) evidence “showed stretching had no effect in reducing injuries.”23 Neither poor quality nor higher quality studies reported any injury prevention effect. Regardless of whether stretching was of individual muscles or entire groups, there was no reduction in injury rates.
More experimental research has been done since. For instance, a 2008 study published in the American Journal of Sports Medicine showed “no significant differences in incidence of injury” in soldiers doing preventative exercises.24 Half of them participated in an exercise program including 5 exercises for strength, flexibility, and coordination of the lower limbs, and 50 of those soldiers sustained overuse injuries in the lower leg, either knee pain or shin splints. The other 500 soldiers were doing nothing at all to prevent injury in the lower limbs — no specific stretching, strengthening or coordination exercises — and only 48 of them had similar injuries. There were “no significant differences in incidence of injury between the prevention group and the placebo group,” and the authors concluded that the exercises “did not influence the risk of developing overuse knee injuries or medial tibial stress syndrome in subjects undergoing an increase in physical activity.”
However, what is clear is that the exercise regimen certainly included static stretching, and it certainly did not work any prevention miracles for some of the most common athletic injuries from the knees down. If stretching performs that poorly in such an experiment, how good can it possibly be at preventing other injuries? Probably not very.
Here in Vancouver — a running Mecca — researchers at Simon Fraser University have done an unusually large study of pre-run stretching, with more than 2700 participants. They found “no statistically significant difference in injury risk between the pre-run stretching and non-stretching groups.”25 Injury rates for all kinds of injuries were the same, with or without stretching. It’s almost as though stretching made no difference at all. But make up your own mind!
I’m never surprised by such findings, because I’ve never heard a sensible explanation for how stretching can generally prevent injury. Usually, advocates have a vague notion that “longer” muscles are less likely to get strained: even if garden-variety stretching made muscles longer (which is doubtful in itself), and even if we knew exactly what kind of stretching to do (we don’t), and even if we had the time to stretch every significant muscle group, the benefits would still be relevant to only a small fraction of common sports injuries. An ankle sprain, for instance, or a blown knee — two of the most common of all injuries — probably have nothing to do with muscle length.
Not convinced yet? A 2014 review of exercise therapy for injury prevention in the British Journal of Sports Medicine was completely negative about stretching26 — even though the authors were obviously a bit too optimistic about everything else!27 “Consistently favourable estimates were obtained for all injury prevention measures except for stretching.” Ouch.
There may be some injury prevention powers to stretching — muscle strains seem like the most likely candidate28 — but probably quite specific and missed by many basic, general pre-event stretching regimens.29 For injury prevention, I can think of Sports Injury Prevention Tips that are probably more effective/efficient than stretching.
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That which prevents injury often also treats it, and vice versa, and that’s the basic rationale for stretching as a treatment for injury — better late than never? People believe that stretching prevents injury, and so they extend that assumption to treatment. Unfortunately, we already know that belief is wrong and stretching does not prevent injury… so it’s unlikely that it can treat it either.
There are some more elaborate rationalizations for stretching as a treatment, chiefly that it corrects something.
Exercise is the closest thing there is to a general miracle drug,3031 and strength training is one of the best types of exercise, practically like magic: healthier and more efficient than most people realize.32 It’s also widely accepted as a major part of injury rehab, and stretching goes along for that ride. Although it’s not taken anywhere near as seriously as strengthening, millions of athletes, trainers, coaches, and healthcare professionals still consider it to be a staple of rehab.
Tedious and specific “corrective” or “therapeutic” exercises are a tired cliché of rehab and physical therapy, both strengthening and stretching. It’s what everyone (athletes and non-athletes alike) imagines their training montage will look like if they are ever hurt.
Unfortunately, corrective exercise isn’t a great reason to build strength, and it’s an even poorer justification for stretching. This style of rehab is largely based on the flawed and even harmful assumption that there is something in-correct about injured patients — fragile, weak, uneven — which must be fixed by sufficiently expert and precise exercise prescription.
For instance, there is often an alleged “imbalance” that consists of both overstretched and weak muscles that need to be toughened and tightened up, and tight and overworked muscles that need to be loosened and stretched.33 It’s surprising how much that single idea has boosted the perceived importance of stretching in the rehab equation.
This kind of thinking has been called the “trap” of corrective exercise, for pros and patients.34 (You can read much more about “the trap” in my main strength training article.)
Stretching obviously doesn’t have much to contribute to load management. At best, it could be considered a form of light exercise that could be used for some stimulation in the early stages… but you could and probably should mostly just stick to dynamic joint mobility drills and very easy strength training instead.
If there’s any other justification for stretching in rehab, it has to live in the much smaller neighbourhood of rehab that isn’t all about load management.
Protecting or restoring range of motion after injury
This is the most reasonable reason to stretch for rehab: not because it helps the injury itself to heal, but to prevent any related loss of range of motion, or to restore it. As explained in detail below, there is no doubt that stretching can improve range of motion. But more ROM does not have clear value in general. Many factors restrict its importance even further in a rehab context.
Stiffness is the sensation that inspires stretching in rehab, but injury stiffness — the stiffness that typically afflicts the joints near an injury — usually has nothing to do with “tight” muscles and tendons. It’s mainly just a neurologically imposed inhibition of movement, a kind of pain, a warning about movement rather than a lack of it. To the extent that it actually does involve any shortening of muscle, it’s probably either minor and temporary and things will get back to normal on their own… or it’s more serious and cannot actually be affected by stretching in any case.35
I think there are some clinical scenarios where a little stretching to maintain or restore post-traumatic ROM is probably worthwhile, but they are minor and rare.
Specific stretching for specific injuries
If not to maintain and restore range of motion, why else would you stretch an injury? What else could stretching do for an injury? Most people imagine that it’s about restoring balance: fixing things that are “too tight” and holding back recovery in some way, probably by constantly irritating the injury. In many cases, this hypothetical restriction is considered the root cause of the injury in the first place, which must be addressed to allow healing to proceed.
Stretching itself is not generally rehabilitative, because a limited range of motion does not usually cause or sustain injuries. Every kind of injury has to be considered separately, because there are all kinds of specific ideas about why one should stretch to fix different injuries. But if you go through a list of injuries and their presumed mechanisms, most cannot be blamed on anything that stretching can fix. Maybe none.
For instance, people believe that the reason they get iliotibial band syndrome, one of the two common kinds of runner’s knee is because the iliotibial band is “too tight.” Unfortunately, that almost certainly isn’t the problem, and the IT band is much too tough a structure to stretch in any case.36
A calf-stretching gadget, one of many on the market that are sold with claims that they can treat plantar fasciitis. Science isn’t so sure.
But the arguments for and against stretching for ITBS are completely different than they are for, say, muscle strains or low back pain. Different kinds of injuries, different arguments about stretching. My plantar fasciitis book goes on for three chapters about the possible role of stretching in treating that condition, but the bottom line is that it either doesn’t work, or not all that well.37
Over time, I will expand on a few other major examples. For now, suffice it to say that
There are a handful that might be in the “promising” category, where it’s probably worth giving it a try. It’s not like stretching is expensive or risky. But even in those cases, it’s always arguable that dynamic joint mobility drills and strengthening would be better choices. For instance, in the case of muscle strain (tear), stretching may help cue the healing mechanisms in your muscle to lay down new connective tissue in a tidy way, and there’s even evidence to support that38 … but so will gentle contractions. Simply using the muscle almost certainly does the same thing, or does it better.
You can go back and forth like this with almost any injury, but the bottom line is that stretching has never been found to make a significant difference in anyone’s rehab.
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Ironically, stretching can actually cause some of the injuries it is thought to prevent or treat. Although stretching is mostly a safe activity, especially if practiced with a modicum of caution, there are (at least) four potential types of stretching hazards:
- Stretching as a warmup may modestly impair athletic performance, as discussed above. Not a huge concern… but not exactly what people want from their stretching either!
- Traumatic injury (sprains and strains) from over-stretching, fairly common in yoga, dance, martial arts, and so on. People just overdo it.
- Traumatic injury to people who should never stretch (but may not know it) because of medical vulnerabilities directly related to flexibility, mainly the hypermobility spectrum disorders and connective tissue diseases (e.g. Ehlers–Danlos syndrome, Marfan syndrome).
- Unexplained body pain may be worsened by attempts to stretch, for a variety of reasons, probably mostly related to aggravating pre-existing conditions.
Hypermobility and Ehlers-Danlos syndrome
Some people are much more flexible than average — pathologically flexible. “Hypermobility spectrum disorders” (HSD) are a group of conditions defined by joint hypermobility — unexplained joint looseness.39 Ehlers–Danlos syndrome (EDS) is a closely related group with known genetic causes40 that includes hypermobility along with fragile tissues that heal poorly, especially skin, with many consequences. All of these conditions together are quite common, and chronic pain is a routine complication.41
Most people don’t need to stretch, but people with HSD/EDS really should not stretch … and they may not know it. HSD/EDS is often undiagnosed and mistreated; it is clinically important and yet often non-obvious. So lots of people are probably suffering without having any idea why or that they shouldn’t be stretching. Consider this story from a 75-year-old reader:
Upon continued strong urging by my physical therapist, I engaged in “glute” and “quad” stretches — quickly to my detriment and horror. MRI survey confirmed gluteal minimus and medius tendinitis and partial thickness tendon tears.After a year of seeking help and ineffective treatment I was just this month diagnosed by a rheumatologist with hypermobility joint syndrome.
This condition does not seem to be on the “radar” of most docs or PT’s. As a kid, I delighted in acrobatics, some said I was “double-jointed”; and in high school and college, modern dance (which included acrobatics). I was still dancing and hiking up until a year ago, but this injury has been devastating!
Now I understand the problem, so my rehab routine is strengthening within a normal range of motion. Feel-good, easy whole-body stretching only. Swimming has been a great help, along with careful exercising with weights at the gym.
It’s awful that this patient, with a long history of hypermobility, was convinced to overstretch by “strong urging” from a healthcare professional. This is a perfect example of clinical ignorance of hypermobility and the consequences of unjustified enthusiasm for stretching. Most people, even healthcare professionals, are simply oblivious to how common and serious hypermobility is — but it’s highly relevant to the value and safety of stretching.
A strong enough stretch can damage any anatomy, obviously — it’s a physical stress. If applied to tissue that is fragile in any way, harm is more likely. Hypermobile tissues are fragile in a way that is quite obviously related to stretching, but there are many other ways for tissues to be vulnerable to physical damage.
For instance, back pain has many possible causes — including a wide variety of possible issues with the spine, many of which could be aggravated by stretching, even severely. And yet people routinely attempt to self-treat back pain with stretching, more or less oblivious to the possibility that it will do more harm than good in some cases.
Here’s the story of my own ill-fated attempt to treat some neck pain with stretching:
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The most flexible athletes are not necessarily the most successful.
Flexibility, by William Sands, p. 389
You don’t hear this argument for stretching as often as you hear the others. And yet it comes up, especially with runners, and with athletes who play sprinty team sports. It’s a common practice to stretch when you’re off the field. The habit is probably usually rationalized as an injury prevention method, but many of those athletes will also insist that it enhances their performance — that the muscles “spring back” from the stretch and make them run faster.42
I’ve already mentioned a huge 2011 scientific review by Kay et al that found “overwhelming evidence” that pre-exercise stretching has “no significant effect.” That was not a surprise. What is a little surprising is that the same review showed the opposite of a benefit — that pre-exercise stretching might reduce muscle strength.43 I wouldn’t take the danger too seriously, but it certainly emphasizes the lack of benefit: if anything, it swings the other way. Yikes!
Similarly, research has shown that stretching does not improve sprinting … but it gets worse. What really happens to your sprint if you stretch first? It turns out that, all other things being equal, the athlete who didn’t stretch is actually going to leave you behind! An Australian research group in Perth did this experiment in early 2009. They rounded up a few athletes and tested their sprinting with and without a stretching regimen between sprints.44 The results of the tests were clear: “There was a consistent tendency for repeated sprint … times to be slower after the static stretching.”
If you want to perform in a sprinty sport, you might not want to stretch right before getting your cleats dirty.
There are many possible mitigating factors here.45 However, the complexities only emphasize the absurdity of the legions of people who have an oversimplified faith that stretching gives them an edge. The evidence is clear that stretching is largely pointless for enhancing performance, and quite possibly worse. Scientific reviews just keep reporting the same conclusion.46 Maybe eventually this news will reach amateur athletes? But don’t hold your breath.
What was that about stretching impairing performance? MOAR SCIENCE
A 2014 test found that a nice pre-run stretch causes “a reduced capacity of the skeletal muscle to produce explosive force.”47 Yikes. It’s not a huge effect, but as Alex Hutchinson put it for Runner’s World, “I can’t see anything good about something that makes me go slower but feel like I’m trying harder.” Indeed, it’s literally the opposite of what people want. Even if the harm is too minor to be of concern, it does underline that stretching is not an effective warm-up.
There are many other studies that have shown static stretch-induced performance impairments, and I’ll summarize some of them in a moment. But why would this happen? It might be purely psychological and neurological — basically just a relatively obvious consequence of a relaxing activity right before a vigorous one. Or stretching could temporarily interfere with normal movement function; virtually any way of measuring it might be impaired in the immediate aftermath of a strong stretch, and how long does that go on? If you do a powerful stretch, it’s actually quite obvious that it takes a few moments to get back to normal after exiting the stretch; we all need to flex and squirm a bit to restore normal movement, and it’s possible or even likely — based on the evidence — that this effect lasts for quite a bit longer than is obvious. Anything more than a couple minutes would be a deal-breaker for me, and probably for most athletes.
Here are some more studies about this crappy effect:
- Musham 201048 — Researchers compared dynamic stretching with “active static stretching” (yep, that acronym is “ASS”) and a control group before sprints of over 20 metres. ASS had no effect one way or the other at first, but turned negative over repeated sprints; 65% of results showed a negative impact by the third sprint. This implies an effect on endurance.
- Wolfe 201149 — The baseline performance (V02 max) of ten elite endurance cyclists was checked on three different occasions. On the second and third occasions, some of them did static stretching first (five stretches, each one held for 30 seconds four times in a row). At the five-minute mark in their workouts, their submaximal VO2 spiked a bit. That sounds good to the untrained ear, but it’s actually a bad thing, and so: “Coaches and highly trained endurance cyclists should exclude static stretching immediately prior to moderate intensity cycling as it reduces acute cycling economy.”
- McHugh 201350 — This was a test of the effect of stretching on hamstring stretches with and without spinal flexion, which adds “neural tension” to the hamstring stretch (by pulling on the spinal cord and big nerve roots and nerves like the sciatic nerve). After stretching with neural tension, strength dropped about 12% — quite a bit actually! The effect of regular hamstring stretching was a mixed bag, with reduced strength at short muscle lengths but boosted at long muscle lengths — a fine example of how generalizations about the effects of stretching are foolhardy.
- Lowery 201451 — Pre-run stretching caused an eight percent drop in performance in a one-mile uphill run. Alex Hutchinson had plenty to say about this one, but here’s the nasty nugget: Yikes!
- Konrad 202052 Punctuating the danger of oversimplification, this review strongly emphasizes that “your mileage may vary” (very close to literally). This review reports a range of both positive and negative effects on performance, depending on what kind of stretching and how performance is measured. But, without a doubt, static stretching can impair performance in some ways in some athletes; the authors reported “detrimental effects in performance variables and metabolic variables.” In general, static stretching seems to more likely to hold you back, whereas dynamic stretching is more likely to be helpful.
None of these needs to be a deal-breaker for anyone who loves stretching. The effects are too minor and complex to be much of a concern. But they do vividly show that basic static stretching — the kind of thing almost all amateur athletes do constantly, and way too many elite athletes too — is definitely not helping consistently/much. And that directly contradicts one of the most classic reasons that people stretch.
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Stretching may not have much of a role in treating or preventing sports injuries, but it is the only way to respond to an acute cramp — which can be injurious. The cramps encountered by most people in a fitness context are exertional or exercise-induced cramps, which is just one of many other kinds of unwanted muscle contractions. We can see how complex muscle physiology is just from the number of ways that it can glitch!
Exercise-induced cramps are intense, painful contractions that usually strike when fatigued and/or overheated. They are most common in the legs, especially the calves and hamstrings, more rarely the quadriceps. Fatigue and heat are major factors, but not dehydration and electrolyte shortage (that’s a myth).53 What actually does cause them is still unclear (shocker), along with much else about them.
Regardless of how cramps work, we have to stretch (or be stretched) when they strike: the urge to pull the other way is irresistible, like jerking your hand away from fire. It feels like we have no choice. Stretching isn’t a “treatment” for cramps per se — it’s more like urgent first aid. The only way to cope with an acute exertional muscle cramp is to directly fight the contraction with stretch.
It’s a benefit of stretching in the same sense that not bleeding is a benefit of bandaids. But why? Does a stretch actually “stop” a cramp? Or does it just make it more tolerable while we wait for it to ease? I don’t think anyone knows the answer to these questions.
Cramp first aid is a legitimate use for stretching, even if it doesn’t actually have anything to do with why people normally stretch.
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A lot of stretching happens in an athletic context for the reasons discussed above, but they all have something in common. The underlying general assumption, almost always unstated, goes like this:
Stretching is a major component of fitness, on par with strength and endurance.
That idea breaks down into specific claims that don’t hold up under scrutiny, but no matter how effective that debunking, almost everyone who likes stretching will continue to assume that it’s different for “serious” athletes or athletes who seem to need more flexibility: gymnastics, dance, martial arts, circus arts, and so on. And yet it’s likely that even for those functional goals stretching is not actually anywhere near as important as we have believed, and maybe not even important at all. If all this information is taken to heart, it should be clear that a “serious” athlete might actually want to avoid stretching. They have a lot of other training to do that is definitely more important — and which will also achieve flexibility.
Until fairly recently, there were few major examples of elite athletes rejecting stretching, but that’s changing. The best recent example I know of is that the Australian Ballet has purged stretching; instead, it’s all about training for strength training throughout the full joint range. The Australian Ballet has written about their experience with this: same or better performance, fewer injuries. Wow.
Dogma is powerful. When there’s a long tradition of doing things a certain way, it can be extremely difficult for people to accept that it might not be necessary. For years, I have been getting cranky email from martial artists, sneering smugly about how I obviously know nothing because, clearly, elite martial artists know that they have to stretch. Maybe. I doubt it, and I think that doubt will be confirmed in time, but it’s officially unknown.
I do know the power of dogma. I know how many times in sports history traditional practices have been overturned and replaced by updated beliefs that were required to break new records.
And in fact I do have some personal experience with martial arts, and I know that not every martial artist is flexible or thinks they need to be. My most memorable example was a grizzled old practitioner of Aikido — the most formidable martial artist I ever met, and also the least flexible. He seemed flexible, but it was all in how he used the rather stunted range of motion he had. He worked within his limitations like an artist — a martial artist — and you would never even have guessed he was so stiff unless you spent time with him in training.54 We never stretched much in that dojo, and an MMA club — with some very competitive members — also never did any stretching.
As the years tick by, I predict that there will be more and more stories about elite athletes who no longer stretch — but still kick ass.
Why is it that many Kenyans don’t stretch? Why was legendary coach Arthur Lydiard not a fan of stretching? Why does Galloway say, “In my experience runners who stretch are injured more often, and when they stop stretching, the injuries often go away”?
Bob Cooper, Runner’s World Magazine55
I am a soccer referee, and mostly by happy accident began substituting what you call “mobilizing” for various stretches prior to my matches, and I find this does an excellent job of stimulating the muscles, whereas after only stretching I still seem to be tight for the first several minutes. Then I read this article, which corroborates what I have found in practice!
Carlos Di Stefano, soccer referee (reader feedback)
In the face of so much discouraging evidence, it makes sense to assume that sport itself provides all the “stretching” one needs. The late Mel Siff:
It is almost heretical to question this stretching doctrine, yet it is important to disclose that there is no research which proves categorically that there is any need for separate stretching sessions, phases or exercises to be conducted to improve performance and safety. To appreciate this fact, it is useful to return to one of the clinical definitions of flexibility, namely that flexibility refers to the range of movement of a specific joint or group of anatomical tissues. Moreover, flexibility cannot be considered separate from other fitness factors such as strength and stamina. There is no real need to prescribe separate stretching exercises or sessions, since logically structured training should take every joint progressively through its full range of static and dynamic movement. In other words every movement should be performed to enhance flexibility, strength, speed, local muscular endurance and skill, so that separate stretching sessions then become largely redundant.
Facts and fallacies of fitness, by Mel Siff, p. 123
Siff’s sensible minimalism — from 1988 — stands in stark contrast to a much more common and marketable “flexibility first” approach, an approach that just happens (coincidence, I’m sure!) to give coaches, trainers and therapists something to be expert about: the idea that athletes must make a point of increasing flexibility first (by whatever stretching method), and then train for the strength and coordination to exploit this marvelous new range of motion. That picture is quite likely to be exactly backwards.
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- Why Do Muscles Feel Stiff and Tight? — Maybe your range of motion is actually limited, or maybe it just feels that way
- A Deep Dive into Delayed-Onset Muscle Soreness — The biology & treatment of “muscle fever,” the deep muscle soreness that surges 24-48 hours after an unfamiliar workout intensity
- The Tyranny of Yoga, Meditation, and Mindfulness — Do you really need to try them? How much do they matter for recovery from conditions like low back pain?
- Strength Training for Pain & Injury Rehab — Why building muscle is easier, better, and more important than you thought, and its role in recovering from injuries and chronic pain
Injuries where stretching might play some role in rehab… or where its role particularly needs debunking:
- plantar fasciitis
- Achilles tendinitis
- muscle strains
- patellofemoral pain
- IT band syndrome
- tennis elbow
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Stretching has an extraordinary reputation for being good for aches and pains, but it’s not clear that stretching deserves this honour.
Stretching as therapy mostly rides on the coat-tails of stretching’s indomitable popularity for other purposes, especially the nearly universally accepted idea that flexibility is a pillar of wellness and fitness, on par with strength and endurance. Unfortunately, that claim does not hold up to scientific scrutiny. All common ideas about the benefits of stretching have been shot down by research over the last twenty twenty years.
But if stiffness is like an itch, stretching is how we want to scratch it! And the symptom of stiffness is thoroughly tangled up with chronic pain. The pleasant sensations of stretching seem directly relevant to the symptom of stiffness, like ice on a burn. It feels inherently valuable to people, and I am not knocking “pleasure”!
But not everything that feels pleasant is actually therapeutic, and there are many, many kinds of pain. Even if stretching is good for some of them, it probably isn’t good for all or even most of them.
So what kind of pain does stretching work for? Any of them? And if so, why? I will have some good things to say about it, but there’s also plenty of debunking ahead.
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The anecdotal evidence that stretching “works” for miscellaneous body pain and stiff and aching muscles is substantial. (So is the anecdotal evidence that it can backfire.) There is also some scientific evidence suggesting that stretching is helpful for common stubborn pain problems, such as neck and back pain,5657 but it’s also a complicated, incomplete, underwhelming mess, like a good 2016 study of stretching for neck pain that had completely underwhelming results.58 Or a 2010 study of exercise for headache confirmed that stretching is much less effective than strength and endurance workouts59 — and yet almost everyone thinks that many headaches are caused by neck tension. Or a 2020 trial of stretching for fibromyalgia that damned stretching with very faint praise60 — a “technical” win for stretching, but not a very satisfying one.
There are also popular stretching tactics for common conditions that are clearly a waste of time. One of the best examples is the idea that tight hamstrings cause back pain, and therefore stretching them is good back pain therapy. But they really don’t, and it’s really not (there’s a whole section about this below). And yet that belief probably accounts for at least 50% of all the stretching people do for back pain!
Another good example is the passionate dedication some runners have to stretching their IT bands — a huge, tendon-like structure on the side of the thigh — to prevent/treat runner’s knee. But IT band “tightness” is not what causes iliotibial band syndrome, and you couldn’t stretch it out even if it did.61
What’s wrong with this picture?
She’s not doing much with this classic IT band stretch. Even if her technique was good. Which it’s not. More about IT band stretching.
Stretching doesn’t seem to come close to “curing” anyone of anything, but darned if it doesn’t sometime seem to “take the edge off” enough to make it worth trying. Consider the trial of stretching for fibromyalgia mentioned above: not impressive results, but technically positive. And when your as desperate as most fibromyalgia patients, any relief is welcome — especially if it’s as cheap and safe as a bit of stretching.
So people in pain stretch, and sometimes they feel better. A little bit. For a while.
People who feel stiff and tight usually assume their range of motion is limited by literally short muscles, but this is rarely the case, despite how it feels. Stiffness isn’t the same as being inflexible; they aren’t even really related.
There are many possible causes of soreness and stiffness that stretching has little to do with: sensitization, and positional cervical cord compression and multiple level radiculopathy, vitamin D and magnesium deficiency, and non-obvious entrapment of nerves (neuropathy) and bloods vessels (claudication). Several hard-to-diagnose diseases can involve long-term excessive aches and pains as a major symptom, such as the hypermobility disorders, facioscapulohumeral muscular dystrophy (FSHD), and multiple sclerosis. And then there’s drug side effects, too!62
That is just a sampling; all of these and more are summarized in 34 Surprising Causes of Pain. If you have pain from any of these sources, stretching is quite unlikely to help. In some cases, it might be useful for symptom control, at best.
Of course, there could be reasons why stretching is good for pain that we don’t understand, or are only just barely starting to understand. In the next sections, I’ll get into some of the causes of stiffness that stretching might be more relevant to: inflammation, trigger points, and contracture. But — spoiler alert — there’s literally not one clear promising example of stretching as effective medicine for anything painful. Not one.
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Why do we so clearly get stiffer as we age? Even in people who have nothing in particular wrong with them, none of the many causes of aches and pains mentioned above?
If there’s one thing most responsible for the feeling of needing to stretch, it’s probably the most common cause of aches and pains: “inflammaging.” Chronic low-grade inflammation that gradually escalates over the years, for all kinds of poorly understood reasons.63 This kind of stiffness is basically a form of mild pain that limits range of motion basically by making it uncomfortable. Our brains are more reluctant to allow free, quick movement of sore tissues. Stiffness is probably mainly a form of inhibition, then.
Unfortunately, systemic inflammation cannot be diagnosed or treated reliably by any means: it is simply too complicated and mysterious. Nevertheless, it is a trendy bogeyman, and allegedly anti-inflammatory diets in particular are extremely popular. But the best defense is simply to be as fit and healthy as possible.
And stretching? That seems like a long shot. But it’s worth discussing, at least.
Stretch your inflammation away?
A 2016 study produced one scrap of evidence that stretching reduces inflammation in connective tissues.64 It’s not clear how much “inflammation in connective tissue” is related to inflammaging — maybe none, maybe lots — but obviously there could be a connection.
We do also have some relevant evidence that inflamed connective tissue is associated with back pain,65 which is of course the epicentre of stiffness as we age (although back pain actually backs off quite a bit on the far side of middle-age).
That evidence is all there is, and it’s too scanty to trust yet. In fact, I am confident that these isolated research clues are probably misleading, and I don’t really buy that stretching reduces inflammation any more than I believe that massage reduces inflammation (a popular idea based on one over-hyped study66). But let’s keep our minds open.
If stretching does help some inflammation resolve, obviously that would be good for us. It wouldn’t even have to be a large or consistent effect (neither are the benefits of pain meds). “Taking the edge off” sometimes would be enough to explain the reputation stretching has for relieving stiffness and soreness.
Chances are strong that inflammaging is a steamroller that stretching cannot really touch, however. There are several plausible mechanisms for inflammaging that are unlikely to be affected by stretching. The best hope is that stretching is somehow mildly anti-inflammatory, regardless of what caused the inflammation, but it’s more likely that fitness is much more “anti-inflammatory” than stretching specifically. Practically any functional stimulation of the same tissue — not just stretching — might have the same modest anti-inflammatory effect.
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There is one kind of soreness that is common and might be responsive to stretching: the stiffness and aching associated with those sensitive spots in muscles commonly known as “muscle knots” or trigger points.
The story goes like this: trigger points are isolated regions of contracted muscle fibres, basically micro cramps. If that’s how they actually work, then stretching might be a viable treatment method. Unfortunately, how they work is highly debatable, and basically unknown.
Stretching as a treatment for trigger points has some expert endorsements. In the weighty text Muscle Pain, researchers Dr. David Simons and Dr. Siegfried Mense wrote that stretching “by almost any means is beneficial.” This depends on a chain of assumptions and theories about how trigger points work: the micro-cramp is metabolically exhausting, like an engine revving in the red, producing waste metabolites that pollute and irritate the surrounding tissues, causing pain and more contraction. In theory, a trigger point cannot burn fuel if it is fully elongated, which would give the energy crisis a chance to abate — a vicious cycle breaker.
If they are right, then stretching works about the same way that stretching out a calf cramp works: you win the tug-of-war with spasming muscle, just on a smaller scale. This sounds great on paper, but there are several major problems in both theory and practice. Simons and Mense also emphasize that it has “not been firmly established” that stretching trigger points is helpful, and that stretch works primarily for “newly activated, single-muscle” trigger points … leaving out a lot of trigger points that are serious problems. There are many circumstances in which you cannot realistically hope to win a tug-of-war with a strong one, because it would be too anatomically awkward and/or too painful.
How can we pull apart a powerful contraction knot — a tiny segment of muscle fibres in full spasm — with anything less than pliers, a vice, and a glass of bourbon? We almost certainly do not have the leverage or pain tolerance required, especially if the muscle fights back with a defensive contraction (which may account for the cases that backfire). That trigger point is like a knot in a bungie cord: all we’re going to do is stretch the hell out of the bungie cord on either side of the knot. If it works at all, it probably mostly only works on the milder cases that don’t matter much in the first place.
And then there’s the possibility that Simons and Mense were just wrong, and a trigger point is not like a tiny cramp at all. If there is no metabolic “revving,” no energy crisis to interrupt by pulling muscle proteins apart like kids fighting on a playground, then it’s back to the drawing board: either stretching doesn’t work at all, or we just have no idea how it works. Which is possible.
This topic is covered in much greater detail (about 10x the length of this section) in my trigger points book.
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There are many common types of pain that stretching might be helpful for, but back pain is the 800-lb gorilla of musculoskeletal medicine, the most ferocious problem that many people face. If stretching wants to be known for being good for pain, it needs to be good for back pain. And people certainly believe it is.67
But it’s both under studied and badly studied. I’ll do my best, but don’t expect much data-driven enlightenment on this topic — that data is just not good enough. For instance, the literature is littered with studies that seem “promising,” but don’t stand up to any scrutiny. Here’s three telling examples:
- A 2010 experiment described in the Archives of Internal Medicine comparing yoga, a stretching class, and an educational booklet.68 This study has been widely reported as “stretching and yoga work,” rather than “stretching and yoga are equally damned here with faint praise.” Not only is it not persuasive, I think it backfires and convinces me that yoga/stretching really didn’t do much.
- In 2014, Chen et al reported “significant” benefit for nurses with back pain who stretched three times per week after work for six months.69 The results are right in line with what people optimistically expect of a stretching habit: that it’s at least as good as popping ibuprofen. Isn’t it great when Science tells us what we already believe? Unfortunately, it’s a terrible study, just complete garbage. I only cited this paper so I could point at it and laugh before pivoting to the disappointing reality.
- In 2019, Pourahmadi et al reviewed 12 studies and reported happy news about “slump stretching” for back pain70 — similar to a seated toe-touch, with some technical refinements. This is a fine example of a not-so-positive-after-all test in two key ways. First, it’s not actually a muscle stretch, it’s literally spinal cord stretching. So this is not a study of anything most people are actually doing. Second, the quality of the evidence they reviewed was “very low.” So it might work… but we have no way to know based on a few scraps of shabby data.
I’ve never yet found a promising study that really was. Meanwhile, there is plenty of actual “evidence of absence.” Three examples:
- In 2014, Sihawong et al proved in a roundabout way that stretching doesn’t prevent back pain, even in people with poor trunk flexibility.71 They studied hundreds of office workers who had no back pain, yet, and followed them for a year while half of did exercises and half didn’t. That’s a fine design in many ways… but they combined stretching with other kinds of exercise that almost certainly account for the entire observed benefit.
- An interesting 2021 study compared stretching unfavourably to a more elaborate exercise therapy, “motor skills training.”72 Stretching lost (and so did strength training). Not by a lot, but it didn’t have to: even with unimpressive competition, stretching was not a contender.
- A 2020 review of studies of exercise for back pain — a good review in British Journal of Sports Medicine — concluded that stretching doesn’t work.73
That’s enough science of general stretching for back pain. Let’s get more specific now.
Stretching the hamstrings to treat back pain
One of the most common specific ideas about the therapeutic value of stretching is that back pain can be beaten by stretching tight hamstrings. It’s the most promising candidate for a specific rationale for stretching to treat back pain. There are others, but this is the big one — if it doesn’t work out, we can mostly ignore the rest.
The relevance of hamstring stretching to back pain is a bit sketchy (understatement). The fear is that tight hamstrings will make your lower spinal joints behave differently and, presumably, suffer greater strain, ultimately succumbing to painful degenerative changes — the kinds of changes that are well-known to be a poor explanation for most back pain, especially in younger people.74 You can’t make back pain with one ingredient any more than you can make a stew with just carrots.
It’s hard to even identify people with tight hamstrings. There are several common simple tests of hamstring flexibility, but they are notoriously unreliable, and there’s really only one method that should be used for research.77 How often has that method actually been used in research? You get a gold star if you guessed “almost never.”
A couple more interesting studies:
- The standing torture test — How long do you think you could stand before your back started to ache? Scientists asked 20 brave volunteers — healthy folks with good backs — to stand for two hours.78 (The inhumanity! Ban human testing!) Exactly half of these folks got back pain during the test. Their hamstring flexibility was measured before and after… and there was no correlation between tight hammies and succumbing to back pain while standing. Hmmm.
- Hamstring tightness and lumbar joint movement — Is there any connection between hamstring flexibility and the amount of lumbar movement when reaching forward? People sure think so, and they’d be right … but only if you’re talking about healthy people. Not so much in back pain patients! A 2010 study was so simple that the results are hard to argue with, even if it’s hard to know what they mean: some low back pain patients use their lumbar joints when they reach, others not so much, and good luck predicting which ones based on hamstring flexibility. You’ll fail if you try, this data says.79 The lack of correlation persisted even after recovery. The point? There’s definitely no clear evidence that lumbar joint movement is wonky in back pain patients with tight hamstrings. Which is really the only plausible mechanism by which it could cause trouble in the first place.
Marshall et al:
With no relationship to actual disability and contradictory findings in the literature for the relationship of the hamstrings to the mechanics of the low back, it is unclear whether decreased hamstring extensibility should be targeted in rehabilitation programs for axial lower back pain.80
All this keeps me from getting excited about the potential of hamstrings or any other stretch target. But, unlike so many other alleged problems, most people can easily fix hamstring tightness. So you might as well! Just don’t waste a lot of time on it: a simple stretching program will work just fine.
Muscle and tendon, although they are distinct tissues, blend together quite seamlessly. Much of what we think of as mucle is an extension of tendinous tissue, and vice versa. It’s impossible to draw a line where tendon stops and muscle starts, and if stretching doesn’t do much to muscles, it probably doesn’t do much to tendons either.
And so most likely a positive effect of stretching on tendons is minimal or nil. Digging a little deeper …
In general, tissues are stimulated to growth and repair by the same forces that they normally have to deal with (and also the same forces that occasionally overload and overwhelm them and cause overuse injury or trauma). That stimulus is dished up far more efficiently and thoroughly by normal (and athletic) activity than by any isolated deliberate exercise therapy.
Cells inside of tendons generate collagenous fibres and absorb others as needed in response to stresses, constantly remodelling and tweaking the tendon so that it is optimized to cope with the actual stresses it encounters all day, every day. (Organisms always act on the assumption that the immediate future will probably be similar to the immediate past — that doesn’t always work out, but it’s a pretty good rule of thumb.)
However, tendons are quite static compared to other tissues, and remodelling is slow and “conservative” — they don’t do it quickly. Even a very strong stretch to a tendon constitutes an extremely brief input of stimulus relative to the context of an entire day or week of normal usage of the tendon. It probably takes months of regular, consistent, and significant new stresses for a tendon to change.
For comparison, consider how bone remodels — and bone is much more dynamic and responsive than tendons are. If bones are subjected to strong new stresses, they will change, slowly but steadily getting thicker and tougher in just the right way to cope with that stress. But it takes a lot! Now, how much do you suppose you could influence that process by deliberately applying a force to the bone? Even a heroic twenty-minute application per day — far more than anyone would ever bother stretching a single tendon, or pair of tendons? And even if it could work, what are the chances that the deliberate application of force would be a good enough “simulation” of natural biomechanical stresses that it would elicit the desired, relevant adaptation? A simulation might be good enough in principle in some cases, but it’s just not much like the stresses that the tendon actually has to deal with in the real world — and therefore it’s a fundamentally inefficient way of preparing for those stresses. If it works at all.
About alignment … the specific notion that tendon stretching will “align” its fibres is a particularly dubious and overly optimistic concept. Tendons are well nigh impervious, rupture only with extreme forces (and/or when already compromised), and change only in response to long term “just right” overloading. It’s relevant to understand that they are so tough that they are the strongest link in the chain, and in many cases they will tear away from their moorings on bone (avulsion fracture) before the “rope” breaks. For a mere stretch, collagen fibres don’t line up obediently any more than they already are — and tendons have impressively well-aligned microscopic orderliness to begin with.
It’s also important to keep in mind that study after study after study has shown no injury prevention benefit to stretching … and that includes tendon injuries. Tendons are not getting injured any less frequently in people who stretch a lot. If you want to reduce the chances of your tendons rupturing, then the way to do it is to expose them to a bunch of activities. Push the envelope just a little: enough that they are challenged, but not brutalized! Just the right amount of stimulation.
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If stretching is mostly irrelevant to pain and injury, why is it that I feel like I have to stretch or I’m going to seize up like an old piece of leather? Why do I have this compulsion to stretch, and why does it feel so good, if it’s not actually doing anything? Why is this true for so many of us?
Because it is probably actually doing something! It’s just probably not doing what you thought it was doing. And we don’t really know for sure what it is doing. If we are intellectually honest, we simply have to admit that.
People routinely report that stretching feels good, that it reduces muscle soreness, or that they feel a strong urge to stretch. And I’m one of them. I have a stretching habit because it feels good, and because it feels like I’m going to “seize up” if I don’t. In particular, I stretch my hamstrings regularly and strongly, and it feels as pleasantly essential to my well-being as slipping into a hot bath — but the exact nature of the benefits are completely unclear to me, and I suspect it’s about as medically useful as a back scratch.
It’s probably a stew of genuine but mysterious and subtle physiological benefits — like the heart rate regulatory effect noted in the last section — plus almost certainly some good placebo, too. I was raised on stretching. Despite my doubt about the conventional wisdom, I tend to emotionally “believe” in stretching just like everyone else — it’s deep in our culture, and, since stretching feels good, it’s easy for my mind to jump to the conclusion that it must be good. But of course that’s not really helpful at all — lots of things feel good without having any clear physiological benefits. Stretching might be like scratching: an undeniably strong impulse, but with almost no relevance to athletic performance or overall health.
I just don’t know. And based on the research to date, no one else does either.
If people believed that feeling good was the only thing that stretching was good for, most people — especially the athletes — would drop it from their exercise routine immediately. Most of us have better things to do. However, if someone firmly declared, “I stretch just to feel good,” I would applaud and say, “Hallelujah! That is an excellent reason to stretch! And one of the few that I can defend!”
And, then again, there may actually be real physiological benefits to stretching — just not the usual ones that get tossed around.
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Is food an effective therapy because it tastes good?
Is music an effective therapy because it sounds good?
Essentially every pleasant sensation and experience has therapeutic qualities. These therapeutic qualities are not unimportant, but they’re not the same thing as an effective therapy. There’s a good reason why your physical therapist never prescribes ice cream. Here’s the last big stretching mystery I’d like to cover: how can stretching be so pleasant without (apparently) doing much measurable good? Look at this pattern:
- Stretching feels great … but it’s over-rated and nowhere near as medically or athletically useful as most people think.
- Massage feels even better … but its effects on pain are notoriously mild and fleeting.
- Chiropractic “adjustments” can feel scrumptious, even addictive, especially in that cinder-block-rigid area between the shoulder blades … but in most cases you’ll be craving a re-do before long (which makes for a lovely business model for chiropractors).
The pattern is that of being “relieved” instead of “fixed.” Over many years of thinking about pain and therapy, it has been a stubborn mystery to me why these things can feel so good — really, really good — without making any large or lasting difference to most painful problems, most of the time.
Feeling good without working all that well causes no end of confusion and trouble. Wonderful and profound sensations are largely responsible for an epidemic of excessive optimism about their healing powers. It’s understandable that we would expect something that feels that good to work well, but a lot of testing has shown over and over again that stretching, massage and chiropractic are not exactly saving the world from its aches and pains.
It’s not hard to explain how something might feel good without curing pain. Sex feels great, but it does not cure pain. Back scratches, chocolate cake, sunshine, and hot baths: all wonderful, all mostly powerless to cure pain.
But stretching is where the gap between how it feels and how well it works is the most glaring, the best feeling but most useless of “treatments.” I’ve already mentioned that I do stretch regularly because I like it, but that doesn’t quite cover it: I actually stretch for pleasure almost every single day. I’m as inflexible and prone to aches and pains as ever, and I’ve never been able to justify the habit with anything except, “Because it feels good, dammit.” And that’s fine.
But why does it feel that good? “It’s stimulating” just doesn’t cut it. I can give a specific reason why each of the delicious things above feel so good. But stretching? I just don’t get it.
And then one day — while I was stretching, of course — I had an epiphany: stretching doesn’t just feel like scratching an itch, maybe it’s actually scratching an itch. A deep itch. In my experience, stretching feels best when I am sore from working out — which only deepens the mystery. Why would it feel so pleasant to pull on soft tissues that are incredibly sore?
That soreness is like an internal “rash” or any skin irritation. And we feel an incredible compulsion to scratch rashes, mosquito bites and other itchy, irritated things. Consider the mosquito bite: scratching it is certainly not going to “treat” it, and we know it. But the temporary relief of scratching is so great it almost transcends pleasure and degenerates into a nasty compulsion. As many pleasures do.
Could it be that exercise-induced soreness is kind of like a minor internal “rash”? And that stretching is just about as close as we can get to “scratching” it? To get a little fleeting relief? This is the best analogy I’ve come up with yet to describe how stretching feels to me.
There may be many reasons why stretching feels good without being particularly helpful, but this makes some serious sense to me. It’s specific and plausible. It achieves the difficult trick of simultaneously accounting for both the unusually pleasant sensation and the more or less total lack of any meaningful effect. And it nicely fits the way I like stretch best when my muscles feel the worst. And it makes even more sense if you extend the metaphor of the itch to include the even more common sensations of being stuck or stagnant, which I’ve written about in the past (guest posting for Todd Hargrove’s excellent blog, see The Bamboo Cage).
I have often said that stretching and other relieving sensations of massage or spinal adjustment feel “like” scratching an itch. But I never went that extra step and considered that maybe they feel like that because, in a way, they actually are — because we can have genuine internal “itches,” vague sensory annoyances … and very limited and indirect ways of scratching them.
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“Contracture” is the unlovely process of muscle and other soft tissues seizing up in response to neurological problems or prolonged immobilization. Your face can’t really “freeze like that” as your mother warned you, but if you could make an ugly face long enough — weeks — eventually contracture really would set in. This is pathology, mind. The tissue changes. For the worse.
Most people probably assume that long, intense stretches must be an effective prevention/treatment for contracture, perhaps the only viable option. It is not an assumption held with much conviction, but stretching always gets the benefit of the doubt, whether it deserves it or not, and it seems to make sense that stretching would be a cure for contracture.
Common sense fails again. As it so often does. The Cochrane Collaboration published a review of static stretch for the treatment and prevention of contractures.81 The verdict? Thumbs way down. Based on “high quality evidence” they concluded that “stretch is not effective for the treatment and prevention of contractures.” I’m shocked. Shocked, I say!
Treatments are usually more obviously valuable to those who need them more. For example, the effect of acetaminophen is more obvious to someone with a headache. This very basic principle doesn’t always apply, but it usually does. This evidence shows that stretch does not meaningfully help even for a condition where the need for tissue elongation is dramatic.
So this is (yet another) great example of a “technical” reason to stretch that many or most people would assume to be effective. But no — probably not static stretch, anyway.82
Pathologically seized up tissue cannot be meaningfully elongated. So what’s happening when healthy people seem to get flexible?
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Some pain is caused by physical interference with nerves — neuropathy. Neural mobilization or neurodynamics is a loose collection of experimental treatments for neuropathy based on the idea that physical stimulation through movement is good for unhappy nerve tissues. But not just any movement: only quite specific movements need apply. If you really know your stuff, you can stretch in a way that emphasizes effects on nerves rather than muscles and tendons. More specifically, it may be possible to slide them in their sheathes (neural gliding or, goofily, neural “flossing”), or just pulling on the whole thing (neural tensioning).
This is a fine example of a specialized type of stretching that might have genuine benefits for some patients. If it works, it would be an impressive partial vindication of therapeutic stretching.
Spoiler alert: no one actually knows if it works.
Nerve stretching just hasn’t been studied well enough. It’s a tough thing to test — technical, messy, hard to standardize — and the science is so threadbare that I’m going to skip citations for this section, other than one review.83 Writing my own informal literature review would just be frustrating and disappointing for everyone, a doomed attempt to pull a useful signal out of a little bit of noise.
So this is not a scientifically validated therapy. But it hasn’t actually been kicked to the curb yet either, it remains plausible and interesting, and no discussion of advanced stretching for pain would be complete without it.
Meet the wire-pipes
“Wiring” seems like the obvious metaphor for nerves, but “plumbing” is surprisingly apt as well. Nerves may not be as much like pipes as blood vessels, but they are more actually more like pipes than wires when it comes to how they work. They are like fancy plumbing doing an impressive imitation of an electrical system.
All nerves are tubes filled with fluid. Although the fluid doesn’t flow, it is wet, which is necessary for all the messy chemistry that allows nerves to play with electricity (just like in batteries). Their clever simulation of a flowing “current” is achieved by ions zipping back and forth across the membrane (action potentials), not by flowing along the length of the nerve (think of a stadium “wave”).
No pipe or wire works like that, of course. These physiology metaphors always fall apart when you start looking too closely.
The chemistry of nerve impulses is sensitive to any interference with the integrity of that membrane. Wires can be kinked like a garden hose with no loss of functionality, but not nerves. Inflammation and other biochemical sources can also mess with nerve function (e.g. several poisons).
Many causes of neuropathy are simple, just straight up pinching. But there are more subtle paths to neuropathy — like not gliding smoothly in their sheaths. Most nerves have a wrapping, but it doesn’t function like the insulation on a wire. Nerves need their own specialized fluid environment for optimal function, so they are a fluid-fluid tube inside another fluid-filled tube. Nested pipes! Exactly like the way the brain and spinal cord are also packed in fluid, just smaller. The whole nervous system is packed in fluid like this, from nerves as thick as your thumb to nerves so tiny they make hairs look like tree trunks.
They are all packed in fluid, and they are all free to slide in those fluid-filled channels to some degree.
Unless they aren’t.
Tunnel syndromes and neural tension: the consequences of nerve snags (from the reasonably certain to the highly speculative)
Sometimes, due to pathological processes, nerves get stuck to the walls of their tubes, like microscopic velcro. This predicament is usually called “neural tension” or a “tunnel syndrome.” You don’t want this happening to your nerves any more than your cat wants tape on its paws. It affects their function.84
There are several infamous and obvious “tunnel syndromes,” most notably carpal tunnel syndrome.85
For every pathologically blatant tunnel syndrome, there are probably many subtler ones. These cases are probably responsible for a lot of milder neuropathy. This is not especially controversial. This scenario is usually called “neural tension” rather than a “tunnel syndrome.”
But what if we ventured even further out onto this limb? What if the most subtle neural stuckness can cause symptoms that would never actually be diagnosed as neuropathy? Just vague aches and twinges and discomfort? Some experts have proposed that these little micro nerve entrapments are actually the specific cause of the “trigger point” phenomenon — humanity’s plague of unexplained sore spots.86 It’s an interesting hypothesis, but that’s all it is so far.
It’s completely unclear whether these subtler scenarios happen at all, and to what degree.
Free the nerves!
You may have guessed it from the setup: the goal of neural gliding is to free the nerves, by stretching in such a way that they actually rip free of their adhesions. Sounds traumatic, but it could be so microscopic that we don’t feel a thing as it happens, or only the normal discomfort of a potent stretch.
Anything that feels like a stretch is undoubtedly causing many, many nerves to slide in their sheaths to some degree, and maybe this is a reason why stretch feels good/healthy: because it breaks up lots of little neural adhesions. Probably not much more than miscellaneous physical activity, and maybe not all, but still, it might be why people like stretching. Just keeping those wire-pipes slip-sliding around nicely!
Neural gliding as a formal therapeutic method is more focused: specific stretches based on detailed anatomical knowledge required to apply maximal tension to specific nerves. There are professionals who have devoted a great deal of energy to learning how to tug on many different nerves. (Heck of a thing, pursuing so much specialized knowledge about a largely unvalidated approach to solving a problem.)
And what about neural “tensioning”?
Neural gliding has the relatively obvious goal of breaking adhesions between nerves and their sheathes. But the point of neural tensioning is just to pull on nerves, without optimizing for “glide” of the nerve within a sheath. That is, some stretches pull on both sheath and nerve more equally.
I am not aware of any specific rationale for just generally stretching nerves, other than the simplistic idea that it’s basically a way of stimulating/exercising them.
Nerve tension and stretch tolerance
Hitting the natural and pathological limits of how far nerves can be pulled is one of the things that determines how intense a stretch feels. We may think we’ve stretched as far as we can not because we’ve hit the mechanical limit of the extensibility of our tissues, but because we’ve moved a nerve as far as it can be moved in its sheath. That may be what causes discomfort when you’re deep in a hard stretch, and it might be why some stretches are more unpleasant to hold than others.
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- Your Back Is Not Out of Alignment — Debunking the obsession with alignment, posture, and other biomechanical bogeymen as major causes of pain
- Does Posture Matter? — A detailed guide to posture and postural correction strategies (especially why none of it matters very much)
- Why Do Muscles Feel Stiff and Tight? — Maybe your range of motion is actually limited, or maybe it just feels that way
- A Deep Dive into Delayed-Onset Muscle Soreness — The biology & treatment of “muscle fever,” the deep muscle soreness that surges 24-48 hours after an unfamiliar workout intensity
- The Tyranny of Yoga, Meditation, and Mindfulness — Do you really need to try them? How much do they matter for recovery from conditions like low back pain?
- Strength Training for Pain & Injury Rehab — Why building muscle is easier, better, and more important than you thought, and its role in recovering from injuries and chronic pain
- Pain is Weird — Pain science reveals a volatile, misleading sensation that is often more than just a symptom, and sometimes worse than whatever started it
- The 3 Basic Types of Pain — Nociceptive, neuropathic, and “other” (and then some more)
- 34 Surprising Causes of Pain — Trying to understand pain when there is no obvious explanation
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There is only one effect of stretching that seems to be almost completely uncontroversial: it does actually increase flexibility. Even plain old “static” stretching, and not even that much of it. Just applying tension to soft tissue will probably do the job.
The phenomenon is widely observed, and seems to have been confirmed by experiments. Many studies over many years have shown it in many ways.8788899091
For whatever it’s worth. But what is it worth, exactly?
The American College of Sports Medicine believes that flexibility is “important in athletic performance (e.g. ballet, gymnastics) and in the ability to carry out activities of daily living.” It’s a “major component” of fitness, along with body composition, cardiovascular endurance, muscle endurance, and muscle strength. They recommend 2–3 days of stretching per week (2–4 repetitions of multiple stretches per day).92
Personal trainers sure buy into it. About 80% of American trainers prescribe static stretching — for all the reasons, but mainly for the flexibility.93
But when you try to spell out exactly why flexibility matters, it’s surprising how quickly things get a bit “um, er, well.” Obviously it’s helpful for some specialized needs — it’s the most important thing on every contortionist’s resumé. But not especially valuable to most athletes. Even many athletes who supposedly need extra flexibility. People love the idea of being flexible, but good luck defining specific benefits for most of them. Read on to watch me try and fail.
It’s possible, but how difficult is it?
Flexibility is not an easy achievement to unlock. It may depend on your genes. I really can’t seem to do it, for instance, which I established long ago with an intense personal experiment. Many other people feel defeated by the challenge as well. Here’s a story sent in by a reader who has been working on it for decades:
I have done yoga every day for more than 20 years. I’m really devoted to it. I do different sets of positions every 2 weeks, but I start my sessions every day with 5-10 minutes of breathing in simplified/easier lotus position (padmasana, where one foot is on the floor, and another is on top of the opposite thigh). It is still just as uncomfortable now as it was when I started. I can handle it for those 5-10 minutes, but more is just too hard. I can still do the full version, but only for a minute or so. One would hope after so many years, doing it daily, I would become more flexible! But not even a little bit — I do the yoga for other reasons.
Franjo M, California
However, for many people, a diligent effort over a period of weeks will probably increase your range of motion. How much of an effort? What “dosage” of stretching is required to get the flexibility effect you want? More on this shortly.
And how does flexibility work?
Once I’ve explored how difficult and pointless increasing flexibility is, I’ll wrap up one of the biggest controversies in stretching science: how the heck does flexibility increase anyway. It’s a matter of nearly pure intellectual curiosity and zero practical significance, but it is fascinating.
When someone increases their flexibility, what exactly changes? How does it work? Turns out flexibility is much more complicated than rocket science. It’s either plasticity or tolerance, or some combination, and it’s an argument that’s been raging for many years. Do we get flexible because tissues actually get physically longer, like pulling taffy? Or is it because we get used to the discomfort and “learn” to be longer, a sensory and neurological stupid-human-trick? Hint: never bet against neurology.
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Beliefs about flexibility and the optimal means of stretching have often proceeded from assumptions that have never been tested and from an almost religious zeal regarding the perceived benefits of stretching by a few.
Flexibility, by William Sands, p. 389
Hold it right there! For how long exactly, though? And how often? How hard? What works best? Is five seconds of light stretch every other day for a couple weeks enough? Probably not. Or should it be five minutes of brutally yanking on your muscles three times a day for three months? That’s probably too much — even if it was definitely more effective, ain’t nobody got time for that. The bottom line:
- no one really knows
- a reasonable amount is probably good enough
- the returns probably diminish steadily as the dosage increases.
The battle of the experts
Here is a vivid example of how poorly understood this all is. This is an excerpt from one of the text books I used in school in the late 90s, a weighty and authoritative tome, a bible of therapeutic exercise (granted, out of date now, but this is the text I originally learned from):
Several authors have suggested that a period of 20 minutes or longer is necessary for a stretch to be effective and increase range of motion when a low-intensity prolonged mechanical stretch is used.
three citations listed, Therapeutic Exercise, 3rd Ed., Kisner/Colby, p157
Twenty minutes?! Almost no one is stretching for that long. No one’s recommending that. And yet “several authors” have found that it is “necessary”? Naturally that prescription is contradicted in other text books, by other experts, not to mention the fact that it’s extremely impractical. Imagine trying to stretch for injury prevention: 20 minutes for each of even just 10 important muscles would be more than three hours. Again, ain’t nobody got time for that.
The idea of a “correct” dosage (or any aspect of technique) is mostly a fantasy. And yet, somewhere between “not nearly enough” and “way too much” there is probably a sweet spot.
In practice, most people stretch only a small selection of tissues quite briefly, seconds only, and erratically. Only the most diligent of us can actually sustain a daily habit for long. We don’t know if that’s enough to achieve the goal. The evidence suggests that holding a stretch for 15 seconds is better than 5… but only a little bit.94
In 2011, a nicely done experiment by Marshall et al showed that regular hamstring stretching substantially increased range of motion in normal university kids.95 Specifically, after “a 4-week stretching program consisting of 4 hamstring and hip stretches performed 5 times per week,” their range increased about 16˚ or 20%. That’s a real result from a sane stretching dosage.
But what if you’re not entirely sane? More extreme goals and methods deserve some focused attention…
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Acrobats, gymnasts, yogis, contortionists, and martial artists have clearly been pushing the limits for centuries, sometimes achieving uncanny mobility. But these are highly motivated athletes with specific and exotic performance goals and stretching regimens that would definitely intimidate the rest of us, and with good reason: they often injure themselves along the way. Indeed, it may even be necessary to injure joints — to traumatize their capsules and ligaments — in order to get them to move that far.
Even these unusual athletes are not chasing flexibility alone, and most athletes have much higher priority training goals. “Athletes usually don’t require extreme ranges of motion,” writes Todd Hargrove,96 “but rather extreme control at the end of relatively normal ranges of motion.”
Fitness and health are not equivalent. You can be fit for a particular athletic pursuit, but that doesn’t mean you are a healthier person: high performance in a narrow category often comes at great costs (such as joint stability). Flexibility is good for a few specialized tasks … and really not much else. It is obviously useful for gymnasts, for instance.
Is this a good idea? This is an “oversplit.” Regularly doing this kind of thing will certainly make a person flexible … but not “healthy.”
Is this child gymnast going to be more flexible? Oh, yes, I think she will be! Will she perform better? Not necessarily: she might be better off doing strength training at the limits of her range — which will not only increase her range, but improve her control at the limits of her range. Is she going to be “healthier” for it? Blatantly not. And she has a middle-aged guy sitting on her back.
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A ballistic stretch is a bouncing stretch: you stretch up the end of your range (take up the “slack”), and then you “bounce” a little beyond that limit. Technically, you’re using momentum to repeatedly, briefly push into “overstretch” — stretching just past the end of your comfortable range of motion. That’s a bunch of words for something quite simple that people do almost unconsciously when stretching.
But, if you believe anything you read about it, overstretch sounds more dangerous than frayed-bungie-cord jumping.
Ballistic stretching is a notorious old-school type of dynamic stretching that is widely believed to be useless and risky — a belief that is rich with irony.
And how did it get that way when there is no compelling evidence or reason to believe that it’s dangerous?97 And when there actually is evidence that it’s just as good as static stretching for increasing flexibility?9899 It’s like the mirror image of a myth!
Many prominent sources warn people not to “bounce” your stretches, but none provide references, because (to the best of my knowledge) there simply are none to cite, no evidence at all that this style of stretching actually causes more injuries. One of the only relevant studies I have ever seen actually found that a large dose of ballistic stretching actually produced much less soreness than static stretching100 (and, yes, stretching can make you sore). All of these citationless warnings are prominent in Google search results in mid-2021:
- Healthline’s “Ballistic Stretching: Is It Safe?” says it “carries a risk of injury” and “generally not recommended.” Vague to the point of meaninglessness. No citations.
- The American Sports and Fitness Association has a blog post called The Dangers of Ballistic Stretching. “Today the very thought makes us cringe.” No citations, just their institutional authority.
- The American Academy of Orthopaedic Surgeons has a guide called “Warm Up, Cool Down and Be Flexible.” They say, “Do not bounce your stretches. Ballistic (bouncy) stretching can cause injury.” No citations — just their institutional authority.
- Well+Good publishes a page called “Stretch Pros Are Begging You To Never, Ever Do Ballistic Stretches—Here’s Why,” which reads: “Stay far, far away from ballistic stretches” seems to be the general consensus among stretch pros, who widely caution against the practice. No citations — just regurgitating expert opinions.
- ShareCare publishes three expert answers to the question, “Why is ballistic stretching bad?” No citations, and every answer is terrible, full of errors and misleading oversimplifications. I will zoom in on one of these next.
Ballistic stretching does not make muscle “fight back”
All three of the “expert” answers on ShareCare say that bouncing provokes reflexive, protective tightening of the muscle, thus “defeating the purpose.” This is incorrect. They are not expert. There is a grain of truth here, but only a grain: there is such a thing as a protective stretch reflex, but it primarily responds to rapid and unexpected stretch in the mid-range. (This is covered more below in the section about how flexibility works.) When you are voluntarily and trivial bouncing a few degrees out of your comfort zone, that’s nothing your body needs to freak out about, and it doesn’t.
I can’t cite an absence of evidence, but search PubMed and see for yourself. But we can easily infer from other evidence that it’s either not happening at all, or barely, or it doesn’t matter — because, as already noted, ballistic stretching increases flexibility just as well as static stretching. If muscles are fighting back, it isn’t getting in the way of the goal, and hitting that barrier is as harmless as the joint reaching it’s natural limits.
Defiant safety message
I know it all seems very “what could possibly go wrong?” Oh noes, you are pushing beyond your limits! Rapidly! In an “uncontrolled” way!
But I just don’t believe that any reasonably cautious person is going to hurt themselves stretching ballistically. “The dose makes the poison.” Too much of anything will hurt you, of course, and there isn’t an exercise in existence that people haven’t overdone. But no ordinary ballistic stretching intensity is hazardous enough to justify all the paranoia.
In fact, if anything, I suspect that some movement at the end of the range is a cautious thing: a way of testing and exploring how the end of the range feels, of generating more raw sensory data for your nervous system to work with.
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“I want to be more flexible,” people say. Even when they have normal range of motion in every joint. What’s this about? Why are people so keen on being bendier? What is it you want to do with that super power?
There are several elastic superheroes. I think this speaks to our genuine craving for greater range of motion. We love that fantasy. But the reality is that hardly anyone actually needs to be more flexible. Most people have a normal range of motion — that’s why it’s normal! Unless you are specifically frustrated because you lack sufficient range of motion in a joint to perform a specific task, you probably don’t need to be more flexible.
What exactly are you planning to do with your flexibility?
Even abnormally poor flexibility is usually no big deal, as long as you are otherwise healthy. There aren’t many functional limitations caused by being super stiff that actually matter much. A couple examples:
- Toe touching is the ultimate in trivial flexibility goals. People can live their whole lives without being able to touch their toes without any practical consequences. Flexibility should only matter if it can help us do something that actually matters to us. For the martial artist, flexibility enables performance goals that martial artists care about! Great! But if a dude doesn’t care if he can touch his toes ... so what?
- Reaching bra hooks actually matters to women, but — unless something is truly wrong with that shoulder, like frozen shoulder — few women are so inflexible that it’s an issue. If it was a problem, they would certainly think it was well worth fixing — et voila, a job for stretching! But it’s both rare and easy to fix: it wouldn’t take much improvement in shoulder ROM.
For stretching with a flexibility goal to matter, we have to identify a common and meaningful functional limitation that can be overcome by increasing range of motion. There are lots of examples that are rare and trivial … or common and trivial … or rare and meaningful … but not both common and meaningful. And so the seemingly worthy goal of better ROM is actually quite marginalized — it doesn’t and shouldn’t matter to most people, most of the time.
If inflexibility is good enough for Eliud Kipchoge, it’s good enough for you. As of 2018, Mr. Kipchoge is the world’s fastest marathoner.101 But the man cannot touch his toes! This is an anecdote from someone who trained with him leading up to when he broke that record:
He was extremely inflexible. After an easy morning run (16km in 1hr10mins), I stretched with the group. Most of them were fairly flexible in the hamstrings (ie. standing straight legged and bending over to touch your toes with your knees straight and being able to touch your toes. Eliud was miles off). He was nowhere near touching his toes! They all found it hilarious that he couldn’t come close to touching his toes.
Matt Fox for SweatElite.co, “Training with Eliud Kipchoge – 5 Things That Surprised Me”
Flexibility and general health
When people have good aerobic capacity — when they are fit enough to efficiently distribute oxygen to all their cells — they are measurably more functional and healthier in all kinds of other ways too. If stretching is an important part of fitness, then it too should be linked to all kinds of other good stuff.
But it’s not. Stretching barely registers any effect on any measure of health and function. More flexible people do not die less or fall down less as they age. They don’t have clearly higher quality of life (a tough one to measure, but it’s been done). They don’t have less back pain or injuries, and in fact they may have more. They don’t have lower blood pressure, resting heart rate, cholesterol — those markers of fitness are much more strongly predicted by weight and endurance.102
Flexibility also isn’t linked to the other components of fitness: flexible people cannot lift more, run farther, or slip through narrower cracks. James Nuzzo: “Absence of correlations between flexibility and other fitness components indicates flexibility is a distinct trait, but not one particularly important for health and function.”
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What actually changes when we become more flexible? Almost everyone — especially non-experts, and that includes almost all clinicians — assumes that the tissue changes. The stuff that’s being stretched must change its properties: it must get longer, softer, more elastic. The muscles and tendons must be different. A few people add “fascia” for buzzword points.
Must this, must that. It sure seems like the tissue must change, but nothing is as obvious as it seems in musculoskeletal and sports medicine, and it’s probably more complicated. Even the stretching researchers and experts have oversimplified. There have been many ideas and many arguments. Here are some of the major specific candidates that have been explored over the years…
Muscles are mostly liquid, and they act like it: they have properties of a liquid (“visco” for viscosity) and a solid (“elasticity”). Viscosity is a fluid’s resistance to changing shape depending on how quickly force is applied to it, while elasticity is how much and how fast a solid returns to normal after being deformed. The idea is that stretching tinkers with these variables.
Stretching may indeed change these properties, but only slightly and temporarily. Significant and lasting changes in viscoelasticity have not been observed.104
Change like a plastic
Connective tissue is tendons and fascia, the sheets of gristle that don’t just wrap muscles like a sausage-wrapping, but subdivide it into compartments, which are in turn subdivided. Every muscle is a honeycomb of fascia, and the stuff blends seamlessly into tendons at the ends of the muscle.
Plasticity is the mirror-image of elasticity: it’s the tendency of a solid to remain in a deformed shape, rather than snap back. We call it plasticity because plastics are the best well-known example a material that can be easily, permanently changed into a new stable shape. There are many different mechanisms for plasticity. And, yes, connective tissue has some plasticity — pull on it hard enough for long enough, you can definitely permanently change it, like pulling taffy.
But doing so usually involves either injury, or extreme patience and persistence — well beyond the reach of most stretching habits.
Connective tissue is a lot tougher than taffy. This is more like pulling steel cable. Like spider silk, many superlatives apply to the properties of connective tissue. The power to deform it in a reasonable time-frame, without injury, has never been convincingly demonstrated in an experiment.
Just add sarcomeres
Sarcomeres are the microscopic engines of muscle, the contractile “links” in the chain of a muscle fibre. It sounds kooky, but we know that animals do in fact grow new sarcomeres when muscle is immobilized in a lengthened position. Strange as it is, this certainly seems like a plausible mechanism for increasing flexibility.
There’s just several problems: The sarcomeres also shorten, so the overall length of the chain is maintained. The muscle also quickly loses the sarcomeres it gained. And sustained immobilization is extremely different from intermittent stretch, and not a very plausible source — but it’s also a hard thing to study (muscle biopsies are serious).
We have a reflex called the “stretch reflex” that kicks in to protect muscles from potentially dangerous elongation, such as you might experience while wrestling. Perhaps this reflex is partly what limits range of motion, and perhaps it can be trained to be less restrictive. Sounds reasonable, no?
But there’s no clear precedent for changing the sensitivity of any reflex — just try not to blink when you get a grain of sand in your eye.106 In any case, the stretch reflex doesn’t generally seem to kick in during slow stretch (no EMG activity at the end of range of motion). It primarily has a braking function: that is, it activates mainly when load is rapidly and significantly increased, something we need to do countless times per day as a part of normal activity. If the stretch reflex applied for a job controlling slow stretches, it wouldn’t get hired. But it’s complicated, and there is some contrary evidence.107
It’s the sensation, stupid! Stretching increases tolerance of stretching
The biomechanical properties of muscle do not seem to change with improvements in flexibility — which seems a bit nuts, and is a bit of a brain teaser. We have some well-established ways of measuring how a muscle responds to elongation, and it’s quite clear that those measurements do not change in people who improve their flexibility — they just go further.
Clearly something’s changing, but the tissue is not the only possibility. So what else is there?
The mind. Seriously.
Well, the brain — which doesn’t much care what your mind thinks. Brains decide when enough is enough, based on how the stretch feels. Your brain commands you to stop when it thinks the stretch is getting dangerous. And how does it enforce that? Inhibition of the muscles pulling you further into it — a kind of self-paralysis.
Yes, this is my second LoTR reference in one section. But who’s counting?
Your brain is the boss of you. The next section will dig deeper into how this actually works, and the evidence for it.
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In the last section, several possible ways that flexibility increases were explored and mostly dismissed, leaving only the sensory theory of flexibility. This is how Cynthia Weppler and S. Peter Magnusson explained it in their 2010 paper, framing it as the last theory standing:
Increases in muscle extensibility observed immediately after stretching and after short-term (3–8 week) stretching programs are due to an alteration of sensation only and not to an increase in muscle length. This theory is referred to as the sensory theory throughout this article because the change in subjects’ perception of sensation is the only current explanation for these results.
Note that phrasing, “the only current explanation.” That’s a Sherlock Holmesian way of putting it: “Once you eliminate the impossible, whatever remains, no matter how improbable, must be the truth.” It’s a strange, cool, and unexpected conclusion … but it also seems to be all we’ve got left, so we should probably take it seriously. And they do present considerable evidence supporting it. I’ll review some interesting highlights.
The wisdom of the body: why we can only stretch so far
Body is not stiff, mind is stiff.
K. Pattabhi Jois
If the sensory tolerance model of flexibility is correct, then it’s “just” the brain that sets the limits of our flexibility. A lot of tissue can probably be stretched much further than we think it can, but the brain decrees: you’re only going to lengthen your muscles so far, period, end of discussion. Any attempt to go further is simply shut down. It’s not a negotiation … at least not in the short term.
Your body probably has excellent reasons for strictly limiting elongation. When a stretch becomes uncomfortable, that’s your nervous system saying, “No way, sister, we don’t go there — we’ve got some sensible and firm safety rules about this.” The overprotective nervous system is a strong theme in neurology generally, and pain science particularly.)
And you can’t overrule your brain. Don’t make the mistake of thinking you could just blast through that barrier with will power. We humans do not control our own brains. Hell, we can barely keep our minds on a leash.108 But we do have some indirect influence, some levers we can pull on — quite literally in the case of stretching.
Stretching is a way of demonstrating to the brain that it’s safe to stretch a little further, just by getting used to the sensations. Familiarity breeds contempt. If your brain is concerned about you — and it always is, often more than you are — what better way to reassure it than to simply spend time at the edges of your range of motion, with nothing bad happening?
It appears that our brains can be tediously convinced to tolerate more elongation of soft tissue. To some degree.
Brain limits everywhere! Tolerance is not just for flexibility
Just as stretching has Team Tolerance, so does athletic performance in general. We are used to thinking in terms of being held back by a variety of physical limits: we can only run so far or jump so high because the spirit is willing, but the flesh is weak. The reality may be that the flesh is not as weak as we thought, that it’s often capable of doing more than we thought possible, and our spirit is irrelevant: it’s the brain that sets the limits.
What I have boiled down to a few words here, Alex Hutchinson explores for an entire book about “the curiously elastic limits of human performance.”
The evidence is now extremely strong that brains play a major role in regulating our effort, progressively shutting us down — for our own good — as we approach the edges of what is safe, rather than what is possible. The brain uses all kinds of sensory cues and biological parameters — heat, thirst, pain — to keep us from exercising ourselves right off a cliff.109
There’s plenty of scientific controversy about this, and there has been ever since it emerged in the 1990s. But most experts accept that the only question is how much brains set limits.
If tolerance is a thing for several other aspects of performance, it probably is for flexibility too. It is part of a broad pattern of evidence converging on the potency of the brain in regulating almost everything.
Can tolerance explain extreme flexibility?
It’s probably a much better way to explain the feats of yogis and martial artists, whose hypermobility might well be dangerously dysfunctional if it were attributable mostly to plastic deformation. If you actually deformed your muscles and tendons enough to really preztel yourself, they might get too long and loose to be useful and functional in general. Tolerance tidily accounts for how they can both allow extreme elongation, while retaining the ability to return to a normal, functional state. It sounds preferable.
Tolerance also doesn’t rule out the possibility of some plastic change at the extremes. More on this below.
Some interesting science experiments
In 2006, Folpp et al did a nice simple experiment: they studied the effects of a month of daily 20-minute hamstring stretches in 20 people.110 The secret sauce was in exactly what they measured at the beginning and the end. They measured how far the hamstrings stretched with a specific force applied, and that barely budged: one degree more movement after a month of stretching. That doesn’t really sound worth it! But, adding more force, the subjects could tolerate eight degrees more stretch. So, to be clear, their hamstring muscles responded to the same force in exactly the same way despite lots of stretching. But if more force was applied, they could go eight degrees further than when they started.
Implication: their muscles didn’t change, but their tolerance did.
In 2014, Blazevich et al tested calf stretching: several people stretched their calf muscles twice per day for three weeks.111 Each session consisted of 4 stretches of 30 seconds. They were compared to people who did not stretch. Measurements included changes in muscle and tendon mechanics, muscle activity, and spinal motoneuron excitability. The non-stretchers didn’t change, while stretch training “elicited a 19.9% increase in dorsiflexion range of motion (ROM) and a 28% increase in passive joint moment at end ROM.” However, the ROM improvement was not explained by changes in the muscle structure:
Increases in end ROM were underpinned by increases in maximum tolerable passive joint moment (stretch tolerance).
Here’s a neat trick: increasing the flexibility of muscles without even stretching them. If you do some upper body stretching, your lower body will get a little more flexible. Not much, but enough to measure in a controlled test.112 But that’s the only effect — which suggests that it’s due to an increase in tolerance of stretch, and that’s all. It’s one of many interesting clues suggesting that stretch tolerance is the secret sauce in flexibility.
And here’s a cool example of how increasing flexibility may be more of a nervous system “hack” than a matter of changing tissue: if you just add some vibration, even already flexible gymnasts can get a surprising boost in flexibility.113114115 Clearly that is a neurological effect on flexibility … and a very cool one. (Vibration is an interesting topic: see Vibration Therapies, from Massage Guns to Jacuzzis.)
This is not all the evidence, not by a long shot — but it’s certainly enough to establish that Team Tolerance isn’t just guessing.
Increased flexibility may simply be an increased tolerance for the discomfort of excessive muscle elongation.
Who disagrees and why? Team Plasticity strikes back (or just flails)
It seems unlikely that Weppler and Magnusson had the last word in 2010, although that paper seems well regarded. The sensory theory of flexibility has held up quite well, and it has many supporters. It is likely to be less wrong116 than anything else.
But Team Plasticity remains substantial and devout, and some of the objections are very “Oh yeah? Is not!” in character.
The next step up is to just cite our ignorance: there might be unconfirmed structural/behavioural changes in the muscle.117 It’s true, there could be — but we also know that there’s not much significant change in the extensibility of tissue to explain. It’s also unlikely that there is some mysterious property of muscle that makes it glow in the dark… because it does not glow in the dark.
The 2011 study of flexibility I cited above is good example of stubbornly defending plasticity in a more scholarly style. Marshall et al clearly showed that stretching increases flexibility: when subjects were stretched with the same force (torque) applied, pushed to the same level of discomfort, they could go 20% farther.118 The data doesn’t say what changed,119 but the authors have a bad case of wishful thinking and followed their data into an overinterpretation, apparently trying to score points for Team Plasticity.120 Because range increased, but pain at the end of the range did not, they strangely concluded that a change in tolerance was not a factor. But stretching farther without hurting more is exactly what we’d expect to see with an increase in (neurological) tolerance. Although it wasn’t measured, it’s safe to assume the subjects’ pain would have been less if stretched only to the end of their original range. So their take just doesn’t make sense to me — it seems like a straightforward failure to understand the concept of sensory tolerance.
I see a lot of this kind of thing from Team Plasticity these days: weak arguments that seem to miss the point. But they are not completely without evidence to support their position. There’s not much, and it mainly seems to be from studies of hardcore stretching, but it’s something: changes in the physical properties of the tissue that we cannot explain. This is the best example I am aware of, and the only one that I consider to be a solid point against Team Tolerance:
Guissard and Duchateau observed lasting changes (a month) in torque/angle curves after five-weeks of calf stretching, five days per week, for 20 minutes121 — a huge dose compared to what is usually used in research and sports, but not necessarily unusual for rehab. We have no idea how the torque/angle curves changed, and all the candidates have major problems (see above). But change they did, and that’s a clear sign that something in that actual tissue was different.
Plasticity has almost been ruled out by a bunch of studies — here’s another fresh one122 — but not completely, and meanwhile neurological adaptation still hasn’t actually been confirmed. It just remains the best non-eliminated explanation.
This whatever-it-is can’t explain the changes observed from more typical stretching regimens. But of course it is possible that flexibility can increase in more than one way.
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How about this? It’s good for your heart!123 “Enhanced vagal modulation.” That’s fairly straightforward good-news science about stretching. (And yet I’ve never heard anyone say they were stretching for heart health.)
It’s not surprising that movement would have some systemic regulatory effects, but it’s nice to see some corroboration of that common sensical notion, and it’s also nice to know that perhaps just stretching did this (to the extent we can learn anything from a single study). If true, it makes for a nice point in favour of a general stretching habit and yoga, “massaging with movement,” and probably even massage itself. It may have a lot to do with why massage feels good.
But stretching is probably not good for the reasons most people are stretching — not much good, anyway, and certainly not much in any ways anyone has figured out how to measure.
Undoubtedly, some specific stretching techniques are good for specific purposes … but quite different from the stretching goals that most people actually have in mind, if they have any clear goals at all. My concern is not that stretching itself is useless, but that people are stretching aimlessly and ineffectively, to the exclusion of evidence-based alternatives, such as a proper warm-up or mobilizations.
I don’t believe that stretching is any more generally useful for people than it is for cats — you do it when you get up in the morning for a few seconds and then you’re off to the sandbox. That feels good — it’s stimulating and enhances your body awareness, it scratches some simple physiological itch, and that’s fine and dandy. But for most people, most of the time? As a time-consuming therapeutic exercise ritual? Stretching simply has a lousy effort-to-reward ratio.
In the next few sections, I will respond to some of the common objections and questions that readers often have.
No one actually knows what a “proper” stretch is. There are no accepted standards in stretching technique (not even close), no method that is clearly superior, no way to know what’s right, no definition of success and no accepted method of achieving it.
The pretentious subtext of all preaching about stretching technique is always the same: stretching is only valuable if you “know what you’re doing.” And so a number of experts stay in business by advocating a stretching method or rationale that seems to trump all the others. Unfortunately, none of them can agree with each other.
The nice thing about standards is that there are so many of them to choose from.
Andrew S. Tanenbaum
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Yes, it is, and yes, it does — but probably not the benefits that people normally attribute to stretching. Even flexibility is suspect.124 Same with qigong, and the martial arts are full of stretching techniques — some of them appropriated from the modern Western fitness tradition, and others inherited from traditional practices. I advocate this kind of stretching elsewhere in my writings. So what’s the difference?
The difference is in intention. The intention of stretching in the context of good qigong, yoga or martial arts is to focus the mind, to stimulate vitality through a combination of mental and physical exercise. The intention is everything — without the intention, you might as well not bother with these activities.
Most westerners stretch without the foggiest notion of this underlying complexity. Stretching is generally stimulating to body awareness, of course: but that awareness is unsophisticated and incidental, rarely involving any insight more complex than “ooh, that muscle sure is sore.” Without education about intent — without a rich philosophical context — the value of stretching in yoga is just as uncertain as it is in any other situation.
And stretching in yoga also involves some risks. Too often people perceive yoga as an entirely wholesome and harmless activity, but in fact over-stretching injuries and muscle strains are actually common. As with dancing or martial arts, there are many ways to hurt yourself practicing yoga.
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We already know that stretching does not do all the basic stuff we used to hope it was doing, especially injury prevention. Is it good for anything else?
There is no known benefit to greater flexibility, except for:
- bragging rights
- dominating Twister tournaments
- making full use of Indian love manuals
In short, stretching appears to be good for … more stretching. Oh, and of course:
- yes, stretching does feel pleasant
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Did you find this article useful? Please support independent science journalism with a donation. See the donation page for more information and options. Why $6?I would rather have a $3 button, but my hands are tied. It’s weird, but $5 online purchases are rejected by credit card companies at an extraordinary rate, because that price point is strongly associated with fraud (specifically with the phenomenon of “card testing” — bad guys testing stolen card numbers with small purchases). And under $5, the fees start to defeat the purpose of the donation. So $6 is the minimum viable price point for a “micro”-transaction.
About Paul Ingraham
I am a science writer in Vancouver, Canada. I was a Registered Massage Therapist for a decade and the assistant editor of ScienceBasedMedicine.org for several years. I’ve had many injuries as a runner and ultimate player, and I’ve been a chronic pain patient myself since 2015. Full bio. See you on Facebook or Twitter.
- A Stretching Experiment — What happens when you stretch your hamstrings intensely for several minutes a day in a steam room?
- The Unstretchables — Eleven muscles you can’t actually stretch hard (but wish you could)
- Stretching Injury — How I almost ripped my own head off! A cautionary tale about the risks of injury while stretching
- Cramps, Spasms, Tremors & Twitches — The biology and treatment of unwanted muscle contractions
- Sports Injury Prevention Tips — A few evidence-based ways to reduce your risk of injury
- Mobilize! — Dynamic joint mobility drills are an alternative to stretching, a way to “massage with movement”
- Basic Self-Massage Tips for Myofascial Trigger Points — Learn how to massage your own trigger points (muscle knots)
- IT Band Stretching Does Not Work — Stretching the iliotibial band is a popular idea, but it’s very hard to do it right, and it’s probably not worth it
- Some other articles that critically examine popular ideas about health: Does Epsom Salt Work? and Water Fever and the Fear of Chronic Dehydration and Does Posture Matter?
Other interesting reading relevant to stretching:
- “What Happens When We Stretch?” An epic illustrated essay about stretching — essentially a comic-book style version of an article a lot like this one. Inspired by it, according to its creator, Stuart Harper.
- “Stretching ‘fails to stop muscle injury’,” News.BBC.co.uk.
- “Reducing risk of injury due to exercise: Stretching before exercise does not help”. This is a superb British Medical Journal editorial, which adds about fifty kilos of credibility to this article.
- “Effects of stretching before and after exercising on muscle soreness and risk of injury: systematic review”. This is the much more technical article, to which the editorial mentioned above is referring.
- “Stretching before exercise: an evidence based approach”. Another more technical article, but also excellent and heavily referenced.
- Published since this, my article A Deep Dive into Delayed-Onset Muscle Soreness — The biology & treatment of “muscle fever,” the deep muscle soreness that surges 24-48 hours after an unfamiliar workout intensity covers the subject of preventing DOMS more thoroughly.
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This article had a long history of unlogged updates — a decade or so — before I finally started logging them in 2015. Updates shown here are the most recent only.
May 3, 2021 — New section: No notes. Just a new chapter. [Updated section: Cramp first aid: at least one undeniable stretching benefit.]
May 3, 2021 — Rebooted: Over the last couple weeks, this article has been upgraded and expanded in many ways. It is now about 33,000 words long, which gives it the dubious distinction of being the first PainSci article to match the length of the shortest PainSci book. Henceforth, I will call this page a “book.”
May — New section: No notes. Just a new chapter. [Updated section: Despite its reputation, ballistic stretching is not actually unsafe or even useless.]
May — Rewritten: Much more and clearer information about other types of stretching, with new references, plus a rewritten explanation of allegedly “advanced” stretching. [Updated section: This isn’t just about static stretching! Types of stretching.]
April — Science update: Added citations about neck pain, fibromyalgia, headache, runner’s knee. [Updated section: Stretching as therapy for miscellaneous pain and stiffness.]
April — Science update: Added two citations to systematic reviews of research on proprioceptive neuromuscular facilitation. [Updated section: This isn’t just about static stretching! Types of stretching.]
April — Completely rewritten: A total reboot of my coverage of the topic of sensory tolerance for stretch. [Updated section: The case for sensory tolerance (and against it) in detail.]
April — New section: A tour of all the major ways that flexibility supposedly works. [Updated section: How does flexibility work?]
April — Major revision: The introduction to flexibility has not been completely re-written, but close: thoroughly revised and tuned to reflect many changes in this part of the article. [Updated section: Stretching for Flexibility: You can probably increase your flexibility by stretching, for whatever it’s worth… but what is it worth?]
April — Major updates: Extensive improvements: more sub-topics, more references, more detail. Also added a good anecdote about stretching too much. [Updated section: Stretching for back pain (especially the hamstrings).]
April — New section: No notes. Just a new chapter. [Updated section: Nerve stretching (neural mobilization, neurodynamics, nerve gliding, nerve flossing, etc).]
Archived updates — All updates, including 22 older updates, are listed on another page. ❐
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- As detailed as this article is, it’s an article about stretching, not flexibility, so I don’t go into any detail about how strength training can improve flexibility. But it does, and my colleague Nick Ng does go into detail: see How to Become Flexible With Strength Training.
- Nuzzo JL. The Case for Retiring Flexibility as a Major Component of Physical Fitness. Sports Med. 2020 May;50(5):853–870. PubMed #31845202 ❐
This is the first of many citations in this article, but it’s also one of the best and most general, and highly recommended reading for professionals. It is like a more scholarly and formal version of this article.
- Some readers will sniff at this and say that CR is still not “advanced” stretching, but it is certainly still widely used, taught, and touted as being better than humble static stretching. I see trainers using it at the gym all the time. Patients can go for physical therapy pretty much anywhere in the world, and there’s a good chance the therapist will do a whole bunch of CR stretching with them, while charging about a buck a minute.
- Azevedo DC, Melo RM, Alves Corrêa RV, Chalmers G. Uninvolved versus target muscle contraction during contract: relax proprioceptive neuromuscular facilitation stretching. Phys Ther Sport. 2011 Aug;12(3):117–21. PubMed #21802037 ❐
This well-planned experiment tested whether or not the contraction component of a contract-relax stretch actually makes a difference, and clearly found that it does not. The researchers compared a normal CR stretch of the hamstring to a modified one without any hamstring contraction (instead, some other “uninvolved, distant” muscle was contracted). The effect of both stretches was the same — “a significant moderate increase in range of motion.” In other words, it didn’t matter if the hamstring was contracted or not — with or without a contraction, the result was the same. This strongly undermines the central claim of CR-PNF stretching.
This is the only trial I'll cite on this topic; the other two citations are both reviews.
- Hill KJ, Robinson KP, Cuchna JW, Hoch MC. Immediate Effects of Proprioceptive Neuromuscular Facilitation Stretching Programs Compared With Passive Stretching Programs for Hamstring Flexibility: A Critically Appraised Topic. J Sport Rehabil. 2017 Nov;26(6):567–572. PubMed #27632820 ❐ Based on just five eligible studies out of 195, this review concluded that there were “consistent findings from multiple low-quality studies that indicate there is no difference…when comparing proprioceptive neuromuscular facilitation [contract-relax] stretching programs to static stretching programs.” Clearly the evidence is limited, but if there is a signal in that mess, it’s awfully weak. The studies they reviewed are almost certainly being produced by people bias in favour of PNF, and yet they still found nothing despite that bias (which inevitably warps studies in subtle ways).
- Lempke L, Wilkinson R, Murray C, Stanek J. The Effectiveness of PNF Versus Static Stretching on Increasing Hip-Flexion Range of Motion. J Sport Rehabil. 2018 May;27(3):289–294. PubMed #28182516 ❐ Another review of just five studies of PNF stretching. In one study, PNF produced better results; in the other four, there was no significant difference. The authors concluded that “both static and PNF stretching effectively increase ROM; however, one does not appear to be more effective than the other.”
- Other major examples include: masseter and temporalis, the suboccipitals, the thoracic paraspinals, latissimus dorsi, the gluteals, the foot arch muscles, and the IT band (of IT band syndrome fame). Some of these muscles can, sort of, be stretched. But all of them are limited to a moderate intensity stretch at best (e.g., the gluteals), in most people, most of the time, using reasonably accessible methods.
- I spent a decade in clinical practice as a massage therapist, routinely asking my patients about their stretching beliefs and habits, among many other things. These days I am a full-time writer on these topics with a global audience, so I hear vastly more about stretching from readers by email than I ever heard from my patients back in the day. I have a great bird’s eye view of popular opinions about stretch. Professionals send me a lot of mail as well, so I get a lot of exposure to their opinions. Are they more educated about stretch? You’d sure hope so, but that hasn’t really been my impression, to be honest — it really seems like everyone is just repeating things they heard other people say. Professionals just hear and repeat even more.
- More evidence on this is coming, but meanwhile there’s an interesting general principle at work here that is worth pointing out: these goals are proven to be impossible either straightforwardly by an evidence of absence, or less obviously but almost as certainly by a preponderance of evidence of minor efficacy. Proof of the existence of a trivial benefit damns with faint praise, and proving that something barely works is literally almost the same as proving that it doesn’t work at all. This is a chronic problem with many allegedly effective treatments and therapies: they may work a little, but they fail to impress. Stretching suffers from this problem in spades.
- Kay AD, Blazevich AJ. Effect of Acute Static Stretch on Maximal Muscle Performance: A Systematic Review. Med Sci Sports Exerc. 2011 Jun 8. PubMed #21659901 ❐ Researchers looked at more than 4500 studies before choosing about 100 to look at more carefully. They found “overwhelming evidence” of “no significant effect,” and that is certainly no surprise for anyone who had been watching stretching science over the years.
- Soligard T, Myklebust G, Steffen K, et al. Comprehensive warm-up programme to prevent injuries in young female footballers: cluster randomised controlled trial. BMJ. 2008;337:a2469. PubMed #19066253 ❐ PainSci #56160 ❐ In 2008, Norwegian researchers compared injuries in over a thousand female footballers who participated in such a warmup for a season, to another several hundred who didn’t. The athletes with the warmup had fewer traumatic injuries, fewer overuse injuries, and the injuries they did have were less severe. Static stretching was not part of the warmup. “Active stretching” was … but “active stretching” is what I would call “mobilizations” — doing moving lunges, for instance — as opposed to the kind of static or passive stretching that most people think of when they think of stretching.
- Soligard T, Nilstad A, Steffen K, et al. Compliance with a comprehensive warm-up programme to prevent injuries in youth football. Br J Sports Med. 2010 Sep;44(11):787–93. PubMed #20551159 ❐ PainSci #54998 ❐
Researchers found that injury rates were significantly lower in soccer (football) teams that diligently performed warmup exercises (“The 11+”, a warmup program recommended by FIFA, which notably does not include stretching). On the one hand, there was not much difference between a little warming up (low participation) and a bit more warming up (average participation). But players and teams that did an especially good job of warming up (“twice as many injury prevention sessions”) got solid results: “the risk of overall and acute injuries was reduced by more than a third among players with high compliance compared with players with intermediate compliance.” That extra enthusiasm went a long way!
- Bizzini, M. Junge, A and Dvorak, J. The 11+ Manual: A Complete warm-up programme to prevent injuries. FIFA Medical Assessment & Research Centre. Publication date unknown. Accessed 2021-05-08. The 11+ warmup does include some “active stretching,” but not much. It also includes this specific clarification: “Why does the 11+ not include stretching exercises? Research has shown that static stretching exercises have a negative influence on muscle performance [more on this soon], and results on the preventive effect of dynamic stretching are inconclusive [more conclusive these days!]. Stretching exercises are not recommended as part of a warm-up programm…”
- Ultimate is a Frisbee team sport, co-ed and self-refereed, with soccer-like intensity and usually the vibe of a good party. Players tend to be jock-nerd hybrids: lots of engineers and scientists. Hippies invented the sport, but have mostly been displaced, and the sport is now relatively mainstream and there’s a pro ultimate league. I’ve been playing since 1997.
A related example
When I was studying to become a massage therapist, our most scientifically literate instructor dared to share a recent research paper with the class (something no other instructor ever did, which is an ugly truth about my training). The study suggested that massage therapy has no effect on the phenomenon of delayed-onset muscle soreness (Tiidus). This was heretical! Most of the class reacted angrily to this attempt to tip over one of the sacred cows of massage therapy, and the hapless instructor was nearly shouted out of the classroom.
What amazed me most about that was that the belief the students were defending was new to them, based only on what they’d heard from instructors in their first year of classes. And yet it was already essential to their self-image as budding health professionals, a “fact” that they planned to use to promote their services someday. It doesn’t take much for dogma to settle in!
This incident made quite an impression on me, and is partly responsible for my own journey from credulity to skepticism.
- Berrueta L, Muskaj I, Olenich S, et al. Stretching Impacts Inflammation Resolution in Connective Tissue. J Cell Physiol. 2016 Jul;231(7):1621–7. PubMed #26588184 ❐ PainSci #52915 ❐
In a previous experiment, these researchers claim to have found evidence that stretching successfully treated inflammation and pain in rats. In this follow-up experiment, they looked more closely at the biology, measuring markers of inflammation: inflammatory lesion thickness, neutrophil count, resolvin (RvD1) concentration. They believe this shows a “direct mechanical impact of stretching on inflammation-regulation mechanisms within connective tissue.” It’s intriguing science for sure, but replication in humans is needed before we get too excited about it.
- Peterson AM, Pedersen BK. The anti-inflammatory effect of exercise. J Appl Physiol. 2005 Apr;98(4). PainSci #52512 ❐
- It’s just one study. We don’t actually know that what Berrueta et al found is for real, and we won’t know until it has been replicated. More importantly, this was a demonstration of an effect on connective tissue, not muscle — and while muscle is full of connective tissue, DOMS is much more of an issue with muscle than connective tissue, and it is far from guaranteed that an effect on connective tissue has anything to do with DOMS.
- Lund H, et al. The effect of passive stretching on delayed onset muscle soreness, and other detrimental effects following eccentric exercise. Scand J Med Sci Sports. 1998 Aug;8(4):216–21. PubMed #9764443 ❐
From the abstract: “There was no difference in the reported variables between experiments one and two. It is concluded that passive stretching did not have any significant influence on increased plasma-CK, muscle pain, muscle strength and the PCr/P(i) ratio, indicating that passive stretching after eccentric exercise cannot prevent secondary pathological alterations.”
- Cheung K, Hume P, Maxwell L. Delayed onset muscle soreness: treatment strategies and performance factors. Sports Med. 2003;33(2):145–64. PubMed #12617692 ❐
From the abstract: “Cryotherapy, stretching, homeopathy, ultrasound and electrical current modalities have demonstrated no effect on the alleviation of muscle soreness or other DOMS symptoms.”
- Weber MD, Serevedio FJ, Woodall WR. The Effects of Three Modalities on Delayed Onset Muscle Soreness. Journal of Orthopaedic & Sports Physical Therapy. 1994;20(5):236–42. PubMed #9512831 ❐
Forty women were subjected to intense training of the biceps and brachilias muscles, until they were sore, and then treated with one of four treatments for DOMS: massage, TENS (electrical stimulation), and ergometry (basically light exercise, to stimulate metabolic activity), and rest. Treatments were given immediately after and then 24 and 48 hours after. There was no difference in the results for any of the women.
- Herbert RD, de Noronha M, Kamper SJ. Stretching to prevent or reduce muscle soreness after exercise. Cochrane Database Syst Rev. 2011;(7):CD004577. PubMed #21735398 ❐
Does stretching help either before or after exercise to reduce soreness? Nope. This large review of eleven small scientific studies, and one huge one, wrapped up with a clear thumbs down:
The evidence from randomised studies suggests that muscle stretching, whether conducted before, after, or before and after exercise, does not produce clinically important reductions in delayed-onset muscle soreness in healthy adults.
The evidence was “low to moderate,” with “moderate to high” risk of bias, which means most of the researchers were probably hoping to find that stretching does help DOMS … but even with that likely bias, they still didn’t find what they were looking for.
The big study was technically positive, finding an average drop in soreness of four points on a 100-point scale, which is basically meaningless. The variation between the results for individuals is undoubtedly greater than that.
- Hart L. Effect of stretching on sport injury risk: a review. Clin J Sport Med. 2005 Mar;15(2):113–113. PubMed #15782063 ❐
OBJECTIVE: Effect of Stretching on Sport Injury Risk: a Review To assess the evidence for the effectiveness of stretching for the prevention of injuries in sports.
DATA SOURCES: MEDLINE (1966 to September, 2002), Current Contents, Biomedical Collection, Dissertation Abstracts, the Cochrane Library, and SPORTDiscus were searched for articles in all languages using terms including stretching, flexibility, injury, epidemiology, and injury prevention. Reference lists were searched and experts contacted for further relevant studies.
STUDY SELECTION: Criteria for inclusion were randomized trials or cohort studies of interventions that included stretching compared with other interventions, with participants who were engaged in sporting or fitness activities. One author identified 361 articles reporting on flexibility, methods and effects of stretching, risk factors for injury, and injury prevention, of which 6 articles fulfilled the inclusion criteria for meta-analysis.
DATA EXTRACTION: Three independent reviewers blinded to the authors and institutions of the investigations assessed the methodologic quality of the studies (100-point scale) and reached consensus on disagreements. Details of study participants, interventions, and outcomes were extracted. Weighted pooled odds ratios were calculated for effects of interventions on an intention-to-treat basis.
MAIN RESULTS: Reduction in total injuries (shin splints, tibial stress reaction, sprains/strains, and lower-extremity and -limb injuries) with either stretching of specific leg-muscle groups or multiple muscle groups was not found in 5 controlled studies (odds ratio [OR] 0.93; 95% CI, 0.78 to 1.11). Reduction in injuries was not significantly greater for stretching of specific muscles (OR, 0.80; CI, 0.54-1.14) or multiple muscle groups (OR, 0.96; CI, 0.71-1.28). Combining the 3 ratings of methodologic quality, median scores were 29 to 60/100. After adjustment for confounders, low quality studies did not show a greater reduction in injuries with stretching (OR, 0.88; CI, 0.67-1.15) compared with high quality studies (OR, 0.97; CI, 0.77-1.22). Stretching to improve flexibility, adverse effects of stretching, and effects of warm up were not assessed by appropriate intervention studies.
CONCLUSION: Limited evidence showed stretching had no effect in reducing injuries.
- Brushøj C, Larsen K, Albrecht-Beste E, et al. Prevention of overuse injuries by a concurrent exercise program in subjects exposed to an increase in training load: a randomized controlled trial of 1020 army recruits. Am J Sports Med. 2008 Apr;36(4):663–670. PubMed #18337359 ❐
- Pereles D, Roth A, Thompson DJ. A Large, Randomized, Prospective Study of the Impact of a Pre-Run Stretch on the Risk of Injury in Teenage and Older Runners. USATF.org. 2011 Jun 15. PainSci #55243 ❐
- Lauersen JB, Bertelsen DM, Andersen LB. The effectiveness of exercise interventions to prevent sports injuries: a systematic review and meta-analysis of randomised controlled trials. Br J Sports Med. 2014 Jun;48(11):871–7. PubMed #24100287 ❐ PainSci #53226 ❐
- They thought strengthening looked good, but based their very positive-sounding conclusions on only four studies of dubious quality and relevance.
- Behm DG, Blazevich AJ, Kay AD, McHugh M. Acute effects of muscle stretching on physical performance, range of motion, and injury incidence in healthy active individuals: a systematic review. Appl Physiol Nutr Metab. 2016 Jan;41(1):1–11. PubMed #26642915 ❐ PainSci #52916 ❐
This wide-ranging review concluded that “studies indicate a 54% risk reduction in acute muscle injuries associated with stretching.” This is at odds even with previous reviews by some of the same key authors. For instance, while McHugh et al were optimistic in 2010, they based their optimism on speculation and characterized the evidence as limited: “For example, there is a good rationale for why stretching could impact the risk of sustaining a muscle strain injury, but the effect of stretching on muscle strain injuries has not been adequately researched in sports with a high incidence of muscle strains.” That was just six years prior, with almost no relevant new evidence in the interim.
Nearly identical evidence. One review calls it inadequate. The other says stretching prevents muscle strain.
So I guess this question is not settled. 😜
- For instance, if stretching reduces the chances of tearing a muscle, you’d need to stretch the muscles most likely to tear, which are the big muscles of the thighs: quadriceps, hamstrings, and adductors (groin). Hamstrings are likely to be included in most stretching regimens. Quadriceps are often included, but not always. Adductors are often missed or given minimal attention. If stretching reduces the risk of muscle strains, you’d have to make a point of stretching all three — and that’s only the most common strain locations.
- Academy of Medical Royal Colleges. Exercise: The miracle cure and the role of the doctor in promoting it. AOMRC.org.uk. 2015 Feb. PainSci #53672 ❐
This is the primary authoritative source of the quote “exercise is the closest thing there is to a miracle cure.”
- Gopinath B, Kifley A, Flood VM, Mitchell P. Physical Activity as a Determinant of Successful Aging over Ten Years. Sci Rep. 2018 Jul;8(1):10522. PubMed #30002462 ❐ PainSci #53004 ❐
If you want to age well, move around a lot!
We already know that physical activity reduces the risk of several of the major chronic diseases and increases lifespan. “Successful aging” is a broader concept, harder to measure, which encompasses not only a reduced risk of disease but also the absence of “depressive symptoms, disability, cognitive impairment, respiratory symptoms and systemic conditions.” (No doubt disability from pain is part of that equation.)
In this study of 1584 older Australians, 249 “aged successfully” over ten years. The most active Aussies, “well above the current recommended level,” were twice as likely to be in that group. Imagine how much better they’ll do over 20 years …
- Research shows strength training is a much more efficient form of exercise than most people realize, and almost any amount of it is much better than nothing. You can gain strength and all its health benefits fairly easily. For more information, see Strength Training Frequency: Less is more than enough: go to the gym less frequently but still gain strength fast enough for anyone but a bodybuilder.
- The “Upper-Crossed Syndrome” (UCS) sounds impressively technical. It is the apex of clinical storytelling about posture in therapy culture, and the prototypical “muscle imbalance” theory, and yet it basically just means slouching: head and shoulders forward, mostly, with several complicated assumptions about its causes and consequences. With UCS, your muscles are presumed to be like dysfunctional ship’s rigging: some are weak and loose (“inhibited”), while others are too strong and tight (“facilitated”). Viewed from the side, you can draw diagonal lines between these groups that cross. (There’s a Lower-Crossed Syndrome as well.) Dr. Janda was a pioneer and did good work with the information he had at the time, but he was probably mostly wrong about UCS; muscle imbalance theories like UCS have not done well over the last couple decades. For more information, see the upper-crossed syndrome section of my posture article.
- Tumminello N, Silvernail J, Cormack B. The Corrective Exercise Trap. Personal Training Quarterly. 2017 Mar;4(1). PainSci #52905 ❐
The authors decisively but gently tip over the most sacred cow of personal training and therapy: corrective exercise, which is grounded in the assumption that there is something in-correct about patients — fragile, weak, or uneven — which must be fixed by sufficiently expert and precise exercise prescription. Exaggerating the importance of defect-correction is actually dangerous for the patient (nocebic): “When clients are told such things about themselves from an authority figure (as they might be during some corrective exercise evaluations), that this potentially makes one’s clients less resilient and more prone to injury and pain.” Translation sans diplomacy: stop #%&^ telling patients they are fragile and weak!
The corrective exercise trap is also about overconfidence in the value of allegedly advanced rehab exercise, when the evidence is overwhelming that corrective exercise is no better than general exercise. “The danger here” — the trap! — “is that many fitness professionals might end up making their training process more about a formalized evaluation procedure and less about good personal training.”
(See more detailed commentary on this paper.)
- If an injury causes more stubborn loss of ROM by the mechanism of contracture or derangement of the bone or cartilage in a joint, stretching is going to be futile.
- Ingraham. IT Band Stretching Does Not Work: Stretching the iliotibial band is a popular idea, but it’s very hard to do it right, and it’s probably not worth it. ❐ PainScience.com. 3664 words.
- Radford JA, Landorf KB, Buchbinder R, Cook C. Effectiveness of calf muscle stretching for the short-term treatment of plantar heel pain: a randomised trial. BMC Musculoskelet Disord. 2007 Apr;8:36. PubMed #17442119 ❐ PainSci #52975 ❐
This trial of stretching for plantar fasciitis made an effort to blind the participants. That is, the 92 study subjects didn’t know what treatment was being tested, reducing the likelihood of bias in reporting on the results. This was achieved by adding sham ultrasound to both the treatment and control groups, a good strategy. After two weeks, unfortunately there was no difference between the groups: “a two-week stretching program provides no statistically significant benefit in ‘first-step’ pain, foot pain, foot function or general foot health compared to not stretching.”
It could have been a larger, longer study, sure, but 92 feet is quite a decent sample size for studies of this kind of thing. And although stretching benefits might ramp up over time, that’s probably wishful thinking, and you’d certainly hope to see clearer signs of progress after two weeks of diligent stretching.
- Malliaropoulos N, Papalexandris S, Papalada A, Papacostas E. The role of stretching in rehabilitation of hamstring injuries: 80 athletes follow-up. Med Sci Sports Exerc. 2004 May;36(5):756–9. PubMed #15126706 ❐
About 40 Greek athletes who stretched more intensively recovered faster than those who stretched more lackadaisically (and the effect is probably not limited to the Hellenic people). How much faster did they recover? They regained their range of motion about 22% sooner (5.6 days instead of 7.3), and their “rehabilitation period” was about 12% shorter (13.3 days instead of 15 days). The researchers reported that this was of “great importance in treating muscle strain injuries.” I’m not quite so thrilled by those numbers: getting back in the game about 36-hours sooner is not nothing, but I wouldn’t call it greatly important. One could easily argue that a wee improvement in healing time isn’t worth the risk of injury, or even the tedious routine of stretching. One could also argue that they really should have compared stretching to other kinds of rehabilitative exercise, and the difference probably would have disappeared.
- HSD is a bucket diagnosis for people with symptomatic hypermobility, but without a clear diagnosis of a connective tissue disorder, like EDS or Marfan syndrome.
- “The essential difference between HSD and hEDS lies in the stricter criteria for hEDS compared to the HSD.” But it’s very tricky, and those criteria are all very new (see the 2017 EDS International Classification). Ironically, as of 2017, the one sub-type of EDS that does not have an identified genetic cause is the hypermobile EDS (hEDS).
- Scheper MC, de Vries JE, Verbunt J, Engelbert RH. Chronic pain in hypermobility syndrome and Ehlers-Danlos syndrome (hypermobility type): it is a challenge. J Pain Res. 2015;8:591–601. PubMed #26316810 ❐ PainSci #52758 ❐ Hypermobility is “highly prevalent among patients diagnosed with chronic pain.”
- There’s actually an entire stretching book that is largely based on this idea — but that book is conspicuously full of armchair science, and no actual evidence that the ideas are true.
- From Kay et al: “The detrimental effects of static stretch are mainly limited to longer durations (≥60 s) which may not be typically used during pre-exercise routines in clinical, healthy or athletic populations. Shorter durations of stretch (<60 s) can be performed in a pre-exercise routine without compromising maximal muscle performance.”
- Beckett JR, Schneiker KT, Wallman KE, Dawson BT, Guelfi KJ. Effects of Static Stretching on Repeated Sprint and Change of Direction Performance. Medicine & Science in Sports & Exercise. 2009 Jan 5.
- The most obvious wrinkle is that the negative impact of static stretching on sprinting might be short term — were the runners slowed down for only ten minutes? Twenty minutes? As much as sixty? Another important consideration is that the potential therapeutic effects of stretching — still remarkably unproven at this late date in history, but still hypothetically possible — could conceivably outweigh the relatively small cost of being slowed down. For instance, if (big if) stretching significantly reduced the risk of a muscle strain, would that risk reduction be worth the cost to sprinting speed? Probably! If.
- Baxter C, Mc Naughton LR, Sparks A, Norton L, Bentley D. Impact of stretching on the performance and injury risk of long-distance runners. Res Sports Med. 2017;25(1):78–90. PubMed #27912252 ❐
- Damasceno MV, Duarte M, Pasqua LA, et al. Static Stretching Alters Neuromuscular Function and Pacing Strategy, but Not Performance during a 3-Km Running Time-Trial. PLoS One. 2014;9(6):e99238. PubMed #24905918 ❐ PainSci #53972 ❐
- Musham C, Hayes PR. Effect of pre-exercise stretching on repeat sprint performance. Br J Sports Med. 2010;44(14). PainSci #54795 ❐
- Wolfe F, Clauw DJ, Fitzcharles MA, et al. Fibromyalgia criteria and severity scales for clinical and epidemiological studies: a modification of the ACR Preliminary Diagnostic Criteria for Fibromyalgia. J Rheumatol. 2011 Jun;38(6):1113–22. PubMed #21285161 ❐
- McHugh MP, Tallent J, Johnson CD. The role of neural tension in stretch-induced strength loss. J Strength Cond Res. 2013 May;27(5):1327–32. PubMed #23439335 ❐
- Lowery RP, Joy JM, Brown LE, et al. Effects of static stretching on 1-mile uphill run performance. J Strength Cond Res. 2014 Jan;28(1):161–7. PubMed #23588487 ❐
- Konrad A, Močnik R, Nakamura M, Sudi K, Tilp M. The Impact of a Single Stretching Session on Running Performance and Running Economy: A Scoping Review. Front Physiol. 2020;11:630282. PubMed #33551850 ❐ PainSci #51822 ❐
- Minetto MA, Holobar A, Botter A, Farina D. Origin and Development of Muscle Cramps. Exerc Sport Sci Rev. 2013 Jan;41(1):3–10. PubMed #23038243 ❐ PainSci #54733 ❐ “Dehydration (and/or cramps, motor unit action potentials, motor neurons electrolyte depletion) often is given as an explanation for muscle cramps occurring in workers and athletes, although this claim is not supported by scientific evidence.” I discuss this at greater length, with more references, in Cramps, Spasms, Tremors & Twitches.
- His wrist ROM was especially memorable: it was like his hands were welded onto the end of his forearms. He once showed me how far he could bend his wrist back, which was not at all. Aikido uses a lot of wrist locks, so his sturdy and inflexible wrists made him almost impervious to those … but with no detectable functional deficit either.
Cooper, Bob. “The Rules Revisited.” Runner’s World. September, 2009. p. 59.
- Ylinen J, Kautiainen H, Wiren K, Hakkinen A. Stretching exercises vs manual therapy in treatment of chronic neck pain: a randomized, controlled cross-over trial. J Rehabil Med. 2007;39(2):126–132. PubMed #17351694 ❐
This examiner-blinded randomized cross-over trial of 125 patients found pretty promising benefits to both stretching exercises and “manual therapy” for a month, and the researchers concluded that “low-cost stretching exercises can be recommended in the first instance as an appropriate therapy intervention to relieve pain, at least in the short-term.” However, there are several reasons not to get too excited about the significance of this study, perhaps the most important of which is simply that showing some improvement over 4 weeks is hardly an impressive therapeutic accomplishment, and if the study had included a control group it might well have revealed that the “therapeutic” effects weren’t much different than the natural course of the condition — i.e. everyone might have gotten better, with and without therapy of any kind.
- Sherman KJ, Cherkin DC, Wellman RD, et al. A Randomized Trial Comparing Yoga, Stretching, and a Self-care Book for Chronic Low Back Pain. Arch Intern Med. 2011 Oct. PubMed #22025101 ❐
This experiment compared the effects of yoga, a normal stretching class, and an educational booklet on chronic low back pain. The primary findings were that both yoga and stretching seemed to be modestly effective, but neither was better than the other. Back in 2005, the same authors got similar results comparing yoga to conventional therapeutic exercise.
The research has been widely reported as “stretching and yoga work,” with a few writers emphasizing that yoga was no better. However, I haven’t seen anyone report that both stretching and yoga are equally damned here with faint praise, and quite possibly illusory praise: the effect size was modest, just 2.5 points on a scale of 11, and some or all that effect may well be attributable to bias and frustrebo (frustrated placebo) caused by a lack of blinding. Subjects deprived of either a lovely stretching or yoga experience may have reported a more negative experience.
I analyze this study in greater detail in my advanced tutorial, Complete Guide to Low Back Pain
- Tunwattanapong P, Kongkasuwan R, Kuptniratsaikul V. The effectiveness of a neck and shoulder stretching exercise program among office workers with neck pain: a randomized controlled trial. Clin Rehabil. 2016 Jan;30(1):64–72. PubMed #25780258 ❐
A trial of stretching for neck pain, specifically “neck and around shoulder stretching exercises two times/day, five days/week during four weeks.” This study had 96 subjects and was well-controlled: stretching was compared to a control group of patients who received only an educational brochure on posture and ergonomics (which isn’t likely to do much, of course). Nevertheless, the control group did improve over three weeks, and while the stretching group improved more they did not improve much more — roughly a point on a 10-point pain scale, for instance. Nothing to write home about. On the bright side, subjects who stretched more did better.
- Ylinen J, Nikander R, Nykänen M, Kautiainen H, Häkkinen A. Effect of neck exercises on cervicogenic headache: a randomized controlled trial. J Rehabil Med. 2010 Apr;42(4):344–9. PubMed #20461336 ❐
- Gómez-Hernández M, Gallego-Izquierdo T, Martínez-Merinero P, et al. Benefits of adding stretching to a moderate-intensity aerobic exercise programme in women with fibromyalgia: a randomized controlled trial. Clin Rehabil. 2020 Feb;34(2):242–251. PubMed #31847574 ❐
This trial compared a program of endurance training with stretching to another program without the stretching. The results are characterized as a win in the abstract, but the fine print —the actual data — is disappointing. This is yet another one of those “technically positive” studies. A few dozen subjects who stretched did get better scores on four different outcome measures. However, the differences were really quite small.
- This is a topic I cover in considerable detail in an excerpt from an entire book about IT band syndrome. See IT Band Stretching Does Not Work: Stretching the iliotibial band is a popular idea, but it’s very hard to do it right, and it’s probably not worth it.
- Some drugs are notorious for causing joint and/or muscle pain as a side effect. (Other kinds of pain are possible too, but are usually more distinctive and readily identified as a drug side effect. But joint and muscle pain are often mistaken for musculoskeletal trouble, and no one suspects the drug.) The usual suspects are the statins (for lowering cholesterol), bisphosphonates (for osteoporosis and Paget’s disease), fluoroquinolones (a class of antibiotics with an extraordinary range of nasty side effects, most notoriously tendinitis), the retinoids (for skin conditions, like Accutane for acne), and Trintellix (an antidepressant). Withdrawal from benzodiazapenes (Valium, Ativan, Xanax, etc) can be extremely uncomfortable and painful; unaware of the risks, many people stop taking them carelessly and suffer quite a bit without ever knowing why. For more detail on some of these, see 34 Surprising Causes of Pain.
How do we get into this mess? Inflammaging correlates with poor fitness and obesity (metabolic syndrome, the biological precursor to diabetes and heart disease). And that, in turn is linked to chronic psychological stress (and of course biological stresses too, like smoking and sleep deprivation). And then there are several other unproven but plausible reasons why inflammation escalates as we age, such as the accumulation of permanent minor infections, lasting collateral damage from past infections (essentially autoimmune disease and allergies that are too subtle or nonspecific to diagnose), and environmental poisons.For a much more thorough exploration of these possibilities, see Chronic, Subtle, Systemic Inflammation: One possible sneaky cause of puzzling chronic pain.
- Berrueta 2016, op. cit.
- Langevin HM, Stevens-Tuttle D, Fox JR, et al. Ultrasound evidence of altered lumbar connective tissue structure in human subjects with chronic low back pain. BMC Musculoskelet Disord. 2009 Dec;10:151. PubMed #19958536 ❐ PainSci #53554 ❐
Researchers measured the thickness of lumbar connective tissues with ultrasound in 60 chronic low back pain patients and 47 health people. The fascia was about 25% thicker in people with back pain, which is quite a bit, and a surprising finding with potentially major — but unknown — clinical significance. The authors suggest that it could be related to “genetic factors, abnormal movement patterns and chronic inflammation.”
This observation has not been reproduced by other researchers, but a follow-up study in 2011 examined the flexibility of the same tissue, and found it was about 20% less in back pain patients: see Langevin for more commentary on the implications of both studies.
- Ingraham. Massage Does Not Reduce Inflammation: The making of a new massage myth from a high-tech study of muscle samples after intense exercise. ❐ PainScience.com. 5631 words.
- Setchell J, Costa N, Ferreira M, Hodges PW. What decreases low back pain? A qualitative study of patient perspectives. Scand J Pain. 2019 07;19(3):597–603. PubMed #31031264 ❐ This survey of 130 Australians showed that about 40% of them believe that “stretching and therapeutic exercise” relieves back pain. I am sure that number varies quite a bit depending on who and how you ask — I wish they’d asked about stretching alone, for instance. But just the fact that stretching is emphasized like it’s the most important example of therapeutic exercise is rather telling, isn’t it? Clearly the faith is real.
- Sherman KJ, Cherkin DC, Wellman RD, et al. A Randomized Trial Comparing Yoga, Stretching, and a Self-care Book for Chronic Low Back Pain. Arch Intern Med. 2011 Oct. PubMed #22025101 ❐
- Chen HM, Wang HH, Chen CH, Hu HM. Effectiveness of a stretching exercise program on low back pain and exercise self-efficacy among nurses in Taiwan: a randomized clinical trial. Pain Manag Nurs. 2014 Mar;15(1):283–91. PubMed #23266331 ❐
- Pourahmadi M, Hesarikia H, Keshtkar A, et al. Effectiveness of Slump Stretching on Low Back Pain: A Systematic Review and Meta-analysis. Pain Med. 2019 02;20(2):378–396. PubMed #30590849 ❐
- Sihawong R, Janwantanakul P, Jiamjarasrangsi W. A prospective, cluster-randomized controlled trial of exercise program to prevent low back pain in office workers. Eur Spine J. 2014 Apr;23(4):786–93. PubMed #24492949 ❐ PainSci #51730 ❐
- van Dillen LR, Lanier VM, Steger-May K, et al. Effect of Motor Skill Training in Functional Activities vs Strength and Flexibility Exercise on Function in People With Chronic Low Back Pain: A Randomized Clinical Trial. JAMA Neurology. 2021 04;78(4):385–395. PainSci #51820 ❐
Got back pain? Trouble bending over? Twisting this-a-way? Flexing that-a-way? Maybe just keep trying! The “just do it” school of therapy. Motor skills training is basically doing movements that are “difficult to perform because of low back pain.” From a certain perspective, you could even call MST a kind of advanced stretching — because it’s focused on pushing beyond movement limitations. (It has a lot in common with the McKenzie mentioned in the previous footnote, actually, with regards to Owen et al.) van Dillen et al presents evidence that it’s a bit more effective than a more conventional stretching program (and combined with strengthening), but only just a bit. Meh.
In addition to damning stretching with a technical loss, and damning MST with a trivial win, this study has some other problems that I go over in my full commentary.
- Owen PJ, Miller CT, Mundell NL, et al. Which specific modes of exercise training are most effective for treating low back pain? Network meta-analysis. Br J Sports Med. 2020 Nov;54(21):1279–1287. PubMed #31666220 ❐ PainSci #51736 ❐
- This is a very complex point that I have substantiated in extreme detail in other places. For one introduction to the topic, see MRI and X-Ray Often Worse than Useless for Back Pain.
- Hori M, Hasegawa H, Takasaki H. Comparisons of hamstring flexibility between individuals with and without low back pain: systematic review with meta-analysis. Physiother Theory Pract. 2019 Jul:1–24. PubMed #31317831 ❐
- Sadler SG, Spink MJ, Ho A, De Jonge XJ, Chuter VH. Restriction in lateral bending range of motion, lumbar lordosis, and hamstring flexibility predicts the development of low back pain: a systematic review of prospective cohort studies. BMC Musculoskelet Disord. 2017 05;18(1):179. PubMed #28476110 ❐ PainSci #52715 ❐
- Ylinen JJ, Kautiainen HJ, Häkkinen AH. Comparison of active, manual, and instrumental straight leg raise in measuring hamstring extensibility. J Strength Cond Res. 2010 Apr;24(4):972–7. PubMed #20300030 ❐
- Raftry SM, Marshall PW. Does a 'tight' hamstring predict low back pain reporting during prolonged standing? Journal of Electromyography and Kinesiology. 2012 Jun;22(3):407–11. PubMed #22397821 ❐
- Johnson EN, Thomas JS. Effect of hamstring flexibility on hip and lumbar spine joint excursions during forward-reaching tasks in participants with and without low back pain. Arch Phys Med Rehabil. 2010 Jul;91(7):1140–2. PubMed #20599054 ❐
- Marshall PW, Mannion J, Murphy BA. Extensibility of the hamstrings is best explained by mechanical components of muscle contraction, not behavioral measures in individuals with chronic low back pain. PM R. 2009 Aug;1(8):709–18. PubMed #19695522 ❐ This was a small trial for cLBP patients, finding no link between hamstring extensibility and disability.
- Harvey LA, Katalinic OM, Herbert RD, et al. Stretch for the treatment and prevention of contractures. Cochrane Database Syst Rev. 2017 Jan;1:CD007455. PubMed #28146605 ❐ PainSci #52742 ❐
- Perhaps there are other methods of stretching that do work for contracture, but the average person has no idea what those are, and therapists scatter in every direction on this point, failing to agree on which “other method” works because there is no evidence base for it — just a whole lot of guessing.
- Basson A, Olivier B, Ellis R, et al. The Effectiveness of Neural Mobilization for Neuro-Musculoskeletal Conditions: A Systematic Review and Meta-Analysis. J Orthop Sports Phys Ther. 2017 Jul:1–76. PubMed #28704626 ❐
The level of evidence for neural mobilization is “largely unknown” according to this review of forty studies (only seventeen with a low risk of bias, and with lots of variety in methods throughout). The results were mixed, and NM notably failed for relatively straightforward peripheral neuropathies like carpal tunnel syndrome — if NM is good for anything, it should be good for that, at least.
On the bright side, according to the paper, the data “reveals benefits of NM for back and neck pain,” but these are complex conditions and notoriously multifactorial, so I remain pessimistic in the absence of more and better data. As the authors understate: “Due to the limited evidence and varying methodological quality, conclusions may change over time.” I’d say it’s so likely that “conclusions may change” that the word “conclusion” isn’t really useful here.
- The membrane of the nerve itself is no longer floating freely, so ions can no longer rush in and out of that section of membrane quite so well. The result: pain, numbness, tingling.
- The tunnel term mainly refers to glaring cases of nerves that have gotten pinched in some of the larger, more obvious nerve sheathes, like the carpal tunnel. The tunnel can get narrowed by various mechanisms, like swelling, and adhesions may also form (basically getting pinched and then also stuck).
- Quintner JL, Bove GM, Cohen ML. A critical evaluation of the trigger point phenomenon. Rheumatology (Oxford). 2015 Mar;54(3):392–9. PubMed #25477053 ❐ Quintner et al. have hypothesized that “irritated peripheral nerve trunks” are “a rich source” of pain, and may be the true cause of trigger points, rather than the more conventional idea that they are “micro cramps.”
- Freitas SR, Mendes B, Le Sant G, et al. Can chronic stretching change the muscle-tendon mechanical properties? A review. Scand J Med Sci Sports. 2018 Mar;28(3):794–806. PubMed #28801950 ❐ “Stretching interventions with 3- to 8-week duration do not seem to change either the muscle or the tendon properties, although it increases the extensibility and tolerance to a greater tensile force.”
- Behm DG, Blazevich AJ, Kay AD, McHugh M. Acute effects of muscle stretching on physical performance, range of motion, and injury incidence in healthy active individuals: a systematic review. Appl Physiol Nutr Metab. 2016 Jan;41(1):1–11. PubMed #26642915 ❐ PainSci #52916 ❐ “All forms of training induced ROM improvements, typically lasting <30 min.”
- Medeiros DM, Cini A, Sbruzzi G, Lima CS. Influence of static stretching on hamstring flexibility in healthy young adults: Systematic review and meta-analysis. Physiother Theory Pract. 2016 Aug;32(6):438–445. PubMed #27458757 ❐ “In all tests, the results favored static stretching compared to control group… In conclusion, static stretching was effective in increasing hamstring flexibility in healthy young adults.”
- Decoster LC, Cleland J, Altieri C, Russell P. The effects of hamstring stretching on range of motion: a systematic literature review. J Orthop Sports Phys Ther. 2005 Jun;35(6):377–87. PubMed #16001909 ❐
Results were inconclusive as to whether or not any particular type of stretching could improve knee ROM. Overall, however, “the evidence appears to indicate that hamstring stretching increase range of motion,” regardless of the technique used. “Appears to indicate” is pretty inconclusive.
- Harvey L, Herbert R, Crosbie J. Does stretching induce lasting increases in joint ROM? A systematic review. Physiother Res Int. 2002;7(1):1–13. PubMed #11992980 ❐ “The results of four 'moderate' quality studies show a convincing effect of stretching in people without functionally significant contracture.”
- American College of Sports Medicine. ACSM’s guidelines for exercise testing and prescription. 10th ed. Philadelphia: Wolters Kluwer; 2018.
- Waryasz GR, Daniels AH, Gil JA, Suric V, Eberson CP. Personal Trainer Demographics, Current Practice Trends and Common Trainee Injuries. Orthop Rev (Pavia). 2016 Sep;8(3):6600. PubMed #27761219 ❐ PainSci #52598 ❐
- Roberts JM, Wilson K. Effect of stretching duration on active and passive range of motion in the lower extremity. Br J Sports Med. 1999 Aug;33(4):259–63. PubMed #10450481 ❐
Another (small) study to determine if stretching for five or fifteen seconds can make a difference in range of motion of the joint. It concluded: “These findings suggest that holding stretches for 15 seconds, as opposed to five seconds, may result in greater improvements in active ROM. However, sustaining a stretch may not significantly affect the improvements gained in passive ROM.” In other words, the returns diminish quickly.
- Marshall PW, Cashman A, Cheema BS. A randomized controlled trial for the effect of passive stretching on measures of hamstring extensibility, passive stiffness, strength, and stretch tolerance. J Sci Med Sport. 2011 Nov;14(6):535–40. PubMed #21636321 ❐
- Hargrove T. Playing With Movement: How to explore the many dimensions of physical health and performance. 1st ed. self-published; 2019. p. 148
- Hard to prove a negative, of course, but if you go looking for data on this and you actually find some, you’re research fu is stronger than mine.
- Covert CA, Alexander MP, Petronis JJ, Davis DS. Comparison of ballistic and static stretching on hamstring muscle length using an equal stretching dose. J Strength Cond Res. 2010 Nov;24(11):3008–14. PubMed #20375742 ❐
This was a comparison of static versus “ballistic” (bouncing) stretching for the hamstrings, showing trivial superiority of static stretching — so trivial that they were basically identical.
- Konrad A, Stafilidis S, Tilp M. Effects of acute static, ballistic, and PNF stretching exercise on the muscle and tendon tissue properties. Scand J Med Sci Sports. 2017 Oct;27(10):1070–1080. PubMed #27367916 ❐ PainSci #51812 ❐
This was a comparison of the effect of a single dose of static stretching, ballistic stretching, or proprioceptive neuromuscular facilitation (PNF) stretching, but very thoroughly measured (several different metrics). The comparison showed “no clinically relevant difference between the stretching groups.”
- Smith LL, Brunetz MH, Chenier TC, et al. The effects of static and ballistic stretching on delayed onset muscle soreness and creatine kinase. Res Q Exerc Sport. 1993 Mar;64(1):103–7. PubMed #8451526 ❐
- 2:01:39, which is 1:18 faster than the previous record, the greatest improvement since 1967. This dude is fast.
- That’s a lot of claims. Rather than citing to support all of them, I’ll just refer you to Nuzzo’s very detailed review, previously cited, which cites many examples and also acknowlgedes some exceptions. The exceptions, however, mostly just “prove the rule.”
- Ch W, Holzman C, Magnusson SP. Increasing muscle extensibility: a matter of increasing length or modifying sensation? Phys Ther. 2010 Mar;90(3):438–49. PubMed #20075147 ❐ PainSci #55283 ❐
- Mizuno T, Matsumoto M, Umemura Y. Viscoelasticity of the muscle-tendon unit is returned more rapidly than range of motion after stretching. Scand J Med Sci Sports. 2011 May. PubMed #21564309 ❐
Participants stretched their calf muscle; at intervals following the stretch, the stiffness of the muscle and the range of motion of the ankle were tested. Although the stiffness of the muscle improved considerably at first, it decreased quickly; however, the range of motion of the ankle significantly increased and was retained for up to 90 minutes. That is, range of motion was retained even after a brief reduction in stiffness subsided, strongly suggesting that ROM is not limited by the viscoelasticity of the tissue.
This study, although small, is consistent with other evidence and the general trend that the impact of stretch is sensory and neurological, not mechanical — i.e. a mild warm-up, basically.
- Brunello E, Reconditi M, Elangovan R, et al. Skeletal muscle resists stretch by rapid binding of the second motor domain of myosin to actin. Proc Natl Acad Sci U S A. 2007 Dec;104(50):20114–9. PubMed #18077437 ❐ PainSci #52656 ❐
While it is well understood that muscles have a “braking” function that kicks in with rapid increases in load, something that occurs constantly in normal activity, but the molecular mechanisms for braking has been slow . This super cool study shed quite a bit of light on that old puzzle of muscle physiology, showing that muscles brake not by “contracting” to resist the rapid elongation, but by building many extra actin-myosin bonds in an instant — becoming stiffer and less extensible in just a few milliseconds, like a rubber band that gets instantly thicker in proportion to how suddenly you pull on it.
This process is also requires very little energy. So the muscle basically solidifies efficiently.
- Optometrists have a machine that blows a puff of air into the eye. I always enjoy the silly challenge of trying to resist blinking. It’s clearly impossible. In general, humans haven’t shown much potential to resist their reflexes. When the doctor bonks your patellar tendon with a rubber hammer, your leg kicks, period — no amount of practice is going to stop that. (Although, to be fair, I’ve never tried really hard.)
- Blazevich AJ, Cannavan D, Waugh CM, et al. Neuromuscular factors influencing the maximum stretch limit of the human plantar flexors. J Appl Physiol (1985). 2012 Nov;113(9):1446–55. PubMed #22923509 ❐ This study reports some apparently reflexive contraction. “Previous research has rarely found a link between EMG activity and range of motion … In contrast, we found a moderate and statistically significant correlation between the angle of onset of muscle activity (but not EMG amplitude) and peak ROM. To our knowledge, this is the first such report…”
- In general, the brain can’t be manipulated simply by wishing, force of will, or a carefully cultivated good attitude. The brain powerfully and imperfectly controls how we experience potentially threatening stimuli, but I’m sorry to report that you do not control your brain. Consciousness and “mind” are by-products of brain function and physiological state. (Deep, eh?) It’s not your opinion of sensory signals that counts, it’s what your brain makes of them that counts — which happens quite independently of consciousness and self-awareness.
- It’s important to keep in mind that the limits aren’t highly flexible because they exist for really excellent reasons. If all the brain limits were suddenly lifted from all the world’s elite and hardcore athletes, half of them would probably be dead or severely injured within a week. It makes complete sense that it’s really hard to convince the brain to be more permissive.
- Folpp H, Deall S, Harvey LA, Gwinn T. Can apparent increases in muscle extensibility with regular stretch be explained by changes in tolerance to stretch? Aust J Physiother. 2006;52(1):45–50.
- Blazevich AJ, Cannavan D, Waugh CM, et al. Range of motion, neuromechanical, and architectural adaptations to plantar flexor stretch training in humans. J Appl Physiol (1985). 2014 Sep;117(5):452–62. PubMed #24947023 ❐
- Behm DG, Cavanaugh T, Quigley P, et al. Acute bouts of upper and lower body static and dynamic stretching increase non-local joint range of motion. Eur J Appl Physiol. 2016 Jan;116(1):241–9. PubMed #26410819 ❐
In this study, range of motion and strength were tested in the upper body after static stretching in the lower body, and vice versa. Passive range of motion was modestly improved, which is nifty, but not active ROM or strength, suggesting that “enhanced stretch tolerance was likely the significant factor,” as opposed to a mechanical or neural drive mechanism.
- Issurin VB, Liebermann DG, Tenenbaum G. Effect of vibratory stimulation training on maximal force and flexibility. J Sports Sci. 1994 Dec;12(6):561–6. PubMed #7853452 ❐
In this 1994 experiment, as described by Sands et al, gymnasts “used a vibrating ring suspended by a cable, in which the foot of the subject was placed while they stretched forward over the raised leg, targeting the hamstrings. The resulting increase in ROM was astonishing. These researchers demonstrated that vibration could enhance flexibility.” The results were replicated by Sands et al in 2006, and Kinser et al in 2008.
- Sands WA, McNeal JR, Stone MH, Russell EM, Jemni M. Flexibility enhancement with vibration: Acute and long-term. Med Sci Sports Exerc. 2006 Apr;38(4):720–5. PubMed #16679989 ❐
This experiment replicated the results of an intriguing 1994 experiment by Issurin et al. Ten highly trained gymnasts did forward splits with or without vibration. They stretched to the point of discomfort for 4 minutes, alternating between each leg, 10 seconds of stretching at a time. Flexibility immediately after stretching with vibration was dramatically greater; the long-term results were less striking.
- Kinser AM, Ramsey MW, O’Bryant HS, et al. Vibration and stretching effects on flexibility and explosive strength in young gymnasts. Med Sci Sports Exerc. 2008 Jan;40(1):133–40. PubMed #18091012 ❐
Replicates the findings of both Issurin and Sands — “simultaneous vibration and stretching may greatly increase flexibility, while not altering explosive strength.”
Calling an idea “less wrong” is a way to acknowledge that an idea is probably not the whole truth while also asserting that it’s closer to it than other ideas. It’s a much more realistic goal than being “right” about anything controversial or at the edges of complex fields of knowledge. We have a shot at reducing inaccuracy and imprecision, but actually eliminating it is nearly impossible — especially in the life sciences, where emergent properties bubble up through thick layers of interacting systems, shaped by countless variables, and about as comprehensible to us as a microwave is to a dog.
The LessWrong blog says that less wrong expresses two important points:
- A humble recognition that no human is ever going to attain perfectly true beliefs and be right about everything. We should always believe that some of our beliefs are mistaken, we just don’t know which ones.
- A bold recognition that notwithstanding the impossibility of being perfectly right, there is still the possibility of being less wrong. Everyone believes false things, but some believe a lot fewer wrong things than others.
- Blazevich 2012, op. cit. This study is an extremely good example of just how absurdly complex all of this is, and specifically it shows that there are variables we still don’t understand. Consider some of their conclusions:
As reported by previous researchers, subjects who were more flexible demonstrated less resistance to the stretch and were able to tolerate a greater stretch load (i.e., greater “passive” joint moment). However, for the first time, we have also shown that more flexible subjects exhibited greater fascicle rotation during stretch and a later onset of reflex muscle activity (i.e., EMG onset) than their less flexible counterparts. Nonetheless, the angle of EMG onset was not correlated with H-reflex amplitude recorded with the muscles held in a stretched position. The present results show significant differences in neural and muscular responses to stretch between the groups.
- Marshall 2011, op. cit.
- This experiment was agnostic about mechanism. It cannot actually settle the bet — it demonstrated only a reduction in stiffness, but not whether it was due to neural or structural adaptations.
- Several things in the paper suggest their bias, but it’s clearest here: although they concede that they “cannot completely rule out volitional stretch tolerance as a possible explanation for changes in extensibility,” they still conclude that “it does seem that hamstring pain elicited during a passive stretch has little involvement in explaining training related improvements.” Really? Sure seems to me like it could!
- Guissard N, Duchateau J. Effect of static stretch training on neural and mechanical properties of the human plantar-flexor muscles. Muscle & Nerve. 2004 Feb;29(2):248–55. PubMed #14755490 ❐
- Konrad A, Tilp M. Increased range of motion after static stretching is not due to changes in muscle and tendon structures. Clin Biomech (Bristol, Avon). 2014 May 9. PubMed #24856792 ❐
Another data point in the plasticity vs. tolerance debate: “The increased range of motion could not be explained by the structural changes in the muscle-tendon unit, and was likely due to increased stretch tolerance possibly due to adaptations of nociceptive nerve endings.”
- Farinatti PT, Brandão C, Soares PP, Duarte AF. Acute effects of stretching exercise on the heart rate variability in subjects with low flexibility levels. J Strength Cond Res. 2011 Jun;25(6):1579–85. PubMed #21386722 ❐
… multiple-set flexibility training session enhanced the vagal modulation and sympathovagal balance in the acute postexercise recovery, at least in subjects with low flexibility levels. … The present results suggest that stretching routines may contribute to a favorable autonomic activity change in untrained subjects.
- Many readers have pointed out that experienced yoga practitioners are flexible, especially the older ones. For instance, T.G. writes, “In my yoga class, the older people are way more flexible than the younger ones.” It may be that training has made them more flexible, but not necessarily. A perpetually neglected explanation for the flexible-yoga-oldster phenomenon is that people with natural gifts of flexibility like the activity and stick with it, because we humans enjoy almost anything we’re good at … and the less flexible tend to drop out. I’m not saying this is how it is — I truly don’t know — just that it’s the kind of explanation that tends to get neglected. We need to consider such counter-intuitive, inside-out explanations that are, perhaps, less comforting to our biases. Even if there is a training effect, it’s extremely likely that it’s only part of the reason those folks are more flexible, and that’s something most “huge fans” of stretching have simply never considered.